L
Lidia Sava
Researcher at University of Catania
Publications - 16
Citations - 963
Lidia Sava is an academic researcher from University of Catania. The author has contributed to research in topics: Congenital hypothyroidism & Thyroid. The author has an hindex of 12, co-authored 16 publications receiving 924 citations. Previous affiliations of Lidia Sava include Université libre de Bruxelles.
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Journal ArticleDOI
The frequency of cold thyroid nodules and thyroid malignancies in patients from an iodine-deficient area
Antonino Belfiore,Giacomo L. La Rosa,Giuseppina Padova,Lidia Sava,Orazio Ippolito,Riccardo Vigneri +5 more
TL;DR: Two analysis of thyroid cancer morbidity in two adjacent areas of Sicily differing in iodine intake indicate that iodine deficiency may be one factor in the development of certain thyroid malignancies in man.
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Thyroid hemiagenesis: prevalence in normal children and effect on thyroid function.
Raffaella Maiorana,Anna Carta,Giuseppina Floriddia,Daniela Leonardi,Massimo Buscema,Lidia Sava,Francesca Calaciura,Riccardo Vigneri +7 more
TL;DR: This study confirms that thyroid hemiagenesis is nearly always due to left lobe defect, and that its prevalence is similar to the cumulative prevalence of thyroid agenesis and ectopia, and is due to thyroid tissue overstimulation by TSH.
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Subclinical hypothyroidism in early childhood: a frequent outcome of transient neonatal hyperthyrotropinemia.
Francesca Calaciura,R. M. Motta,Giuseppe Miscio,Graziella Fichera,Daniela Leonardi,Anna Carta,Vincenzo Trischitta,Vittorio Tassi,Lidia Sava,Riccardo Vigneri +9 more
TL;DR: Frequent thyroid morphology abnormalities and frequent thyroperoxidase and TSH receptor gene sequence variations were observed, and newborns classified false positive at congenital hypothyroidism screening have a very high risk of subclinical hypothyrogenism in infancy and early childhood.
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Risk factors for congenital hypothyroidism: results of a population case-control study (1997-2003).
Emanuela Medda,Antonella Olivieri,Maria Antonietta Stazi,Michele E. Grandolfo,Cristina Fazzini,Mariangiola Baserga,Massimo Burroni,Emanuele Cacciari,Francesca Calaciura,Alessandra Cassio,Luca Chiovato,Pietro Costa,Daniela Leonardi,Maria Martucci,Lidia Moschini,Severo Pagliardini,Giuseppe Parlato,Alberto Pignero,Aldo Pinchera,Danielle Sala,Lidia Sava,Vera Stoppioni,Francesco Tancredi,Fabiola Valentini,Riccardo Vigneri,Mariella Sorcini +25 more
TL;DR: The results suggested a multifactorial origin of CH in which genetic and environmental factors play a role in the development of the disease.
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Solitary Autonomously Functioning Thyroid Nodules and Iodine Deficiency
TL;DR: Iodine deficiency is one possible factor in the development of autonomously functioning thyroid nodules (AFTN) and that iodine deficiency may also be involved in the increased frequency of toxic evolution of these lesions.