M
M. Ross Bullock
Researcher at VCU Medical Center
Publications - 36
Citations - 3287
M. Ross Bullock is an academic researcher from VCU Medical Center. The author has contributed to research in topics: Traumatic brain injury & Brain damage. The author has an hindex of 27, co-authored 36 publications receiving 3121 citations. Previous affiliations of M. Ross Bullock include University of California, Davis & Veterans Health Administration.
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Journal ArticleDOI
Factors affecting excitatory amino acid release following severe human head injury
M. Ross Bullock,Alois Zauner,John J. Woodward,John S. Myseros,Sung C. Choi,John D. Ward,Anthony Marmarou,Harold F. Young +7 more
TL;DR: The authors evaluated the role of EEAs in human traumatic brain injury in patients who have suffered severe head injury by placing a microdialysis probe into the gray matter along with a ventriculostomy catheter or an intracranial pressure monitor for 4 days.
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Predominance of cellular edema in traumatic brain swelling in patients with severe head injuries
Anthony Marmarou,Stefano Signoretti,Panos P. Fatouros,Gina Portella,Gunes A. Aygok,M. Ross Bullock +5 more
TL;DR: The brain swelling observed in patients with TBI appears to be predominantly cellular, as signaled by low ADC values in brain tissue with high levels of water content.
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Brain oxygenation and energy metabolism: part I-biological function and pathophysiology.
TL;DR: To understand cerebral oxygenation, it is important to understand cerebral blood flow, energy production, ischemia, acidosis, generation of reactive oxygen species, and mitochondrial failure, which provide the basis of knowledge regarding brain bioenergetics and are important topics to understand when developing new approaches to patient care.
Journal ArticleDOI
Guidelines for the Surgical Management of Traumatic Brain Injury Author Group
M. Ross Bullock,Randall M. Chesnut,Jamshid Ghajar,David Gordon,Roger Härtl,David W. Newell,Franco Servadei,Beverly C. Walters,Jack E. Wilberger +8 more
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Anatomical integration of newly generated dentate granule neurons following traumatic brain injury in adult rats and its association to cognitive recovery.
TL;DR: The time course of cognitive recovery following lateral fluid percussion injury in rats and the proliferative response, maturational fate and integration of newly generated cells in the DG demonstrate the extensive anatomical integration of new born dentate granule neurons at the time when innate cognitive recovery is observed.