M
Marco Canepa
Researcher at University of Genoa
Publications - 153
Citations - 3049
Marco Canepa is an academic researcher from University of Genoa. The author has contributed to research in topics: Heart failure & Medicine. The author has an hindex of 26, co-authored 110 publications receiving 2060 citations. Previous affiliations of Marco Canepa include Johns Hopkins University & National Institutes of Health.
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Journal ArticleDOI
Linking Heart Failure to Cancer: Background Evidence and Research Perspectives.
TL;DR: In this article, the authors investigated the link between heart failure and malignancy and found that low-grade inflammation plays a primary role in the development of malignancies in patients with preexisting heart failure.
Journal ArticleDOI
Arterial stiffness and vitamin D levels: the Baltimore longitudinal study of aging.
Francesco Giallauria,Yuri Milaneschi,Yuri Milaneschi,Toshiko Tanaka,Marcello Maggio,Marco Canepa,Palchamy Elango,Carlo Vigorito,Edward G. Lakatta,Luigi Ferrucci,James B. Strait +10 more
TL;DR: Vitamin D levels are inversely associated with increased arterial stiffness in a normative aging population, irrespective of traditional risk factor burden.
Journal ArticleDOI
Characteristics, treatments and 1-year prognosis of hospitalized and ambulatory heart failure patients with chronic obstructive pulmonary disease in the European Society of Cardiology Heart Failure Long-Term Registry
Marco Canepa,Ewa Straburzyńska-Migaj,Jarosław Drożdż,Carla Fernandez-Vivancos,Jose Manuel Garcia Pinilla,Noemi Nyolczas,Pier Luigi Temporelli,Alexandre Mebazaa,Mitja Lainscak,Cécile Laroche,Aldo P. Maggioni,Massimo F Piepoli,Andrew J.S. Coats,Roberto Ferrari,Luigi Tavazzi +14 more
TL;DR: The characteristics and assess the 1‐year outcomes of hospitalized (HHF) and chronic (CHF) heart failure patients with chronic obstructive pulmonary disease (COPD) enrolled in a large European registry between May 2011 and April 2013 are described.
Journal ArticleDOI
p38 MAPK and JNK antagonistically control senescence and cytoplasmic p16INK4A expression in doxorubicin-treated endothelial progenitor cells.
Paolo Spallarossa,Paola Altieri,Chiara Barisione,Mario Passalacqua,Concetta Aloi,Giuseppina Fugazza,Francesco Frassoni,Marina Podestà,Marco Canepa,Giorgio Ghigliotti,Claudio Brunelli +10 more
TL;DR: It is demonstrated that Cord Blood Endothelial Progenitor Cells exposed to low, sub-apoptotic doses of doxorubicin show a senescence phenotype characterized by increased SA-b-gal activity, decreased TRF2 and chromosomal abnormalities, enlarged cell shape, and disarrangement of F-actin stress fibers accompanied by impaired migratory ability.
Journal ArticleDOI
Low Sensitivity of Bone Scintigraphy in Detecting Phe64Leu Mutation-Related Transthyretin Cardiac Amyloidosis
Maria Beatrice Musumeci,Francesco Cappelli,Domitilla Russo,Giacomo Tini,Marco Canepa,Agnese Milandri,Rachele Bonfiglioli,Gianluca Di Bella,Filomena My,Marco Luigetti,Marina Grandis,Camillo Autore,Stefano Perlini,Federico Perfetto,Claudio Rapezzi +14 more
TL;DR: The sensitivity of bone scintigraphy (DPD and HMDP) in detecting TTR-CA is extremely low in patients with Phe64Leu TTR mutation, suggesting the need to identify cardiac involvement across a wider spectrum of TTR mutations.