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Marco Lenzi

Researcher at University of Bologna

Publications -  141
Citations -  13113

Marco Lenzi is an academic researcher from University of Bologna. The author has contributed to research in topics: Autoimmune hepatitis & Hepatitis C. The author has an hindex of 43, co-authored 134 publications receiving 11722 citations.

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Nonalcoholic fatty liver, steatohepatitis, and the metabolic syndrome

TL;DR: The presence of multiple metabolic disorders is associated with a potentially progressive, severe liver disease and the increasing prevalence of obesity, coupled with diabetes, dyslipidemia, hypertension, and ultimately the metabolic syndrome puts a very large population at risk of forthcoming liver failure in the next decades.
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The NAFLD fibrosis score: A noninvasive system that identifies liver fibrosis in patients with NAFLD

TL;DR: A simple scoring system accurately separates patients with nonalcoholic fatty liver disease with and without advanced fibrosis, rendering liver biopsy for identification ofAdvanced fibrosis unnecessary in a substantial proportion of patients.
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Nonalcoholic fatty liver disease a feature of the metabolic syndrome

TL;DR: It is concluded that NAFLD, in the presence of normoglycemia and normal or moderately increased body weight, is characterized by clinical and laboratory data similar to those found in diabetes and obesity.
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Early occurrence and recurrence of hepatocellular carcinoma in HCV-related cirrhosis treated with direct-acting antivirals

TL;DR: In patients with HCV-related cirrhosis, DAA-induced resolution of HCV infection does not seem to reduce occurrence of HCC, and patients previously treated for HCC have still a high risk of tumour recurrence in the short term, despite DAA treatment.
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Type 2 autoimmune hepatitis and hepatitis C virus infection.

TL;DR: The prevalence of serum antibodies to hepatitis C virus (HCV) was assessed by an enzyme-linked immunosorbent assay in 46 patients seropositive for liver-kidney microsomal antibody (anti-LKM1), the marker of autoimmune hepatitis type 2, with similar prevalence to that reported in patients with chronic non A, non B posttransfusion hepatitis.