M
Maria Eugenia Guicciardi
Researcher at Mayo Clinic
Publications - 52
Citations - 5444
Maria Eugenia Guicciardi is an academic researcher from Mayo Clinic. The author has contributed to research in topics: Apoptosis & Liver injury. The author has an hindex of 27, co-authored 52 publications receiving 4927 citations. Previous affiliations of Maria Eugenia Guicciardi include University of Modena and Reggio Emilia.
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Journal ArticleDOI
Apoptosis as a mechanism for liver disease progression.
TL;DR: Therapy focused on interrupting the cellular mechanisms linking apoptosis to fibrosis would be useful in human liver diseases.
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Calpains can do it alone: implications for cancer therapy.
TL;DR: Mu-Calpain Activation in Beta-Lapachone-Mediated Apoptosis results inappa-like responses in mice are consistent with that of a drug-naive animal and suggest that this “drug-free” approach toApoptosis may be a viable alternative for humans.
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COX‐2 inhibits Fas‐mediated apoptosis in cholangiocarcinoma cells
Ugochukwu C. Nzeako,Maria Eugenia Guicciardi,Jung Hwan Yoon,Steven F. Bronk,Gregory J. Gores +4 more
TL;DR: It is suggested that prostanoid generation by COX‐2 specifically inhibits Fas‐mediated apoptosis, likely by up‐regulating Mcl‐1 expression.
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Myofibroblast-derived PDGF-BB promotes hedgehog survival signaling in cholangiocarcinoma cells
Christian D. Fingas,Steven F. Bronk,Nathan W. Werneburg,Justin L. Mott,Maria Eugenia Guicciardi,Sophie C. Cazanave,Joachim C. Mertens,Alphonse E. Sirica,Gregory J. Gores +8 more
TL;DR: MFB‐derived PDGF‐BB protects CCA cells from TRAIL cytotoxicity by a Hh‐signaling–dependent process, and in a rodent CCA in vivo model, cyclopamine administration increased apoptosis in C CA cells, resulting in tumor suppression.
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Caveolin-1 Can Regulate Vascular Smooth Muscle Cell Fate by Switching Platelet-Derived Growth Factor Signaling From a Proliferative to an Apoptotic Pathway
Timothy E. Peterson,Maria Eugenia Guicciardi,Rajiv Gulati,Laurel S. Kleppe,Cheryl S. Mueske,Martina Mookadam,Grzegorz Sowa,Gregory J. Gores,William C. Sessa,Robert D. Simari +9 more
TL;DR: It is suggested that caveolin-1 is an inhibitor of PDGF proliferative responses and might be capable of transforming PDGF-induced proliferative signals into death signals.