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Marina Camatini

Researcher at University of Milano-Bicocca

Publications -  130
Citations -  4370

Marina Camatini is an academic researcher from University of Milano-Bicocca. The author has contributed to research in topics: Cytoskeleton & Immunoelectron microscopy. The author has an hindex of 36, co-authored 130 publications receiving 3838 citations. Previous affiliations of Marina Camatini include University of Perugia & University of Milan.

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Impact of tire debris on in vitro and in vivo systems

TL;DR: The amount of Zn leached from TD is related to pH, aggregation of particles and elution process, the quantity of TD present in the environment has to be taken into account and the atmospheric conditions, which may deeply influence the particle properties, have to be considered.
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Differences in cytotoxicity versus pro-inflammatory potency of different PM fractions in human epithelial lung cells.

TL;DR: The results underline that the in vitro responsiveness to PM may be cell line dependent and suggest that the PM different properties may trigger different endpoints such as inflammation, perturbation of cell cycle and cell death.
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Seasonal variations in chemical composition and in vitro biological effects of fine PM from Milan

TL;DR: The different seasonal chemical composition of PM showed to influence some biological properties, which were investigated by bivariate analysis and multivariate data analysis to investigate the relationship between PM chemical composition and the biological effects produced by cell exposure to 12 microg cm(-2).
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Cell cycle alterations induced by urban PM2.5 in bronchial epithelial cells: Characterization of the process and possible mechanisms involved

TL;DR: Both PM-induced DNA damage and G2 arrest were inhibited by the addition of antioxidants and α-naphthoflavone, suggesting the involvement of ROS and reactive electrophilic metabolites formed via a P450-dependent reaction.
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Airborne urban particles (Milan winter-PM2.5) cause mitotic arrest and cell death: Effects on DNA, mitochondria, AhR binding and spindle organization

TL;DR: It is hypothesized that the induced mitotic arrest and following cell death was due to a premature chromosome condensation caused by a combination of DNA, mitochondrial and spindle damage.