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Maurizio Gualtieri

Researcher at ENEA

Publications -  70
Citations -  3202

Maurizio Gualtieri is an academic researcher from ENEA. The author has contributed to research in topics: Particulates & A549 cell. The author has an hindex of 28, co-authored 64 publications receiving 2539 citations. Previous affiliations of Maurizio Gualtieri include University of the Littoral Opal Coast & Polaris Industries.

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Impact of tire debris on in vitro and in vivo systems

TL;DR: The amount of Zn leached from TD is related to pH, aggregation of particles and elution process, the quantity of TD present in the environment has to be taken into account and the atmospheric conditions, which may deeply influence the particle properties, have to be considered.
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Economic losses due to ozone impacts on human health, forest productivity and crop yield across China

TL;DR: Using O3 exposure-response functions, the costs of O3-induced losses in rice, wheat, forests and forest production, and SOMO35-based morbidity for respiratory diseases and non-accidental mortality are evaluated, representing 7% of the China Gross Domestic Product in 2015.
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Seasonal variations in chemical composition and in vitro biological effects of fine PM from Milan

TL;DR: The different seasonal chemical composition of PM showed to influence some biological properties, which were investigated by bivariate analysis and multivariate data analysis to investigate the relationship between PM chemical composition and the biological effects produced by cell exposure to 12 microg cm(-2).
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Cell cycle alterations induced by urban PM2.5 in bronchial epithelial cells: Characterization of the process and possible mechanisms involved

TL;DR: Both PM-induced DNA damage and G2 arrest were inhibited by the addition of antioxidants and α-naphthoflavone, suggesting the involvement of ROS and reactive electrophilic metabolites formed via a P450-dependent reaction.
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Airborne urban particles (Milan winter-PM2.5) cause mitotic arrest and cell death: Effects on DNA, mitochondria, AhR binding and spindle organization

TL;DR: It is hypothesized that the induced mitotic arrest and following cell death was due to a premature chromosome condensation caused by a combination of DNA, mitochondrial and spindle damage.