M
Marta Murgia
Researcher at Max Planck Society
Publications - 63
Citations - 6330
Marta Murgia is an academic researcher from Max Planck Society. The author has contributed to research in topics: Skeletal muscle & Myosin. The author has an hindex of 30, co-authored 58 publications receiving 5793 citations. Previous affiliations of Marta Murgia include University of Padua & University of Geneva.
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Microdomains with high Ca2+ close to IP3-sensitive channels that are sensed by neighboring mitochondria
TL;DR: It is concluded that in vivo, domains of high [Ca2+]i are transiently generated close to IP3-gated channels and sensed by nearby mitochondria; this may provide an efficient mechanism for optimizing mitochondrial activity upon cell stimulation.
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Chimeric green fluorescent protein as a tool for visualizing subcellular organelles in living cells
TL;DR: GFP is an invaluable new tool for studies of molecular biology and cell physiology as a marker of transfection in vivo and provides a simple means of identifying genetically modified cells to be used in physiological studies.
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Oxidized ATP. An irreversible inhibitor of the macrophage purinergic P2Z receptor.
TL;DR: In this paper, the effects of oxidized ATP on responses triggered by extracellular adenosine 5'-triphosphate (ATPe) were investigated in the mouse macrophage-like cell line J774.
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Mitochondrial Ca2+ homeostasis in intact cells.
TL;DR: A general scheme can be envisaged by which the triggering of a plasma membrane receptor coupled to InsP3 generation raises the Ca2+ concentration both in the cytoplasm and in the mitochondria, where it activates the metabolic activity according to the increased cell needs.
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Calcineurin controls nerve activity-dependent specification of slow skeletal muscle fibers but not muscle growth
Antonio L. Serrano,Marta Murgia,Giorgia Pallafacchina,Elisa Calabria,Patrizia Coniglio,Terje Lømo,Stefano Schiaffino +6 more
TL;DR: The activation of MyHC-slow induced by direct electrostimulation of denervated regenerating muscle with a continuous low frequency impulse pattern is blocked by CsA, showing that calcineurin function in muscle fibers and not in motor neurons is responsible for nerve-dependent specification of slow muscle fibers.