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Paola Pizzo

Researcher at University of Padua

Publications -  120
Citations -  9861

Paola Pizzo is an academic researcher from University of Padua. The author has contributed to research in topics: Endoplasmic reticulum & Mitochondrion. The author has an hindex of 51, co-authored 113 publications receiving 7742 citations. Previous affiliations of Paola Pizzo include National Research Council & Weizmann Institute of Science.

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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Chimeric green fluorescent protein as a tool for visualizing subcellular organelles in living cells

Rosario Rizzuto, +4 more
- 01 Jun 1995 - 
TL;DR: GFP is an invaluable new tool for studies of molecular biology and cell physiology as a marker of transfection in vivo and provides a simple means of identifying genetically modified cells to be used in physiological studies.
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Mitofusin 2 ablation increases endoplasmic reticulum–mitochondria coupling

Riccardo Filadi, +7 more
- 28 Apr 2015 - 
TL;DR: It is demonstrated that Mfn2 ablation increases the structural and functional ER–mitochondria coupling and strengthens the efficacy of inositol trisphosphate (IP3)-induced Ca2+ transfer from the ER to mitochondria, sensitizing cells to a mitochondrial Ca2- overload-dependent death.
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Modulation of the endoplasmic reticulum–mitochondria interface in Alzheimer’s disease and related models

Louise Hedskog, +17 more
- 07 May 2013 - 
TL;DR: It is revealed that nanomolar concentrations of amyloid β-peptide increased inositol-1,4,5-triphosphate receptor and voltage-dependent anion channel protein expression and elevated the number of ER–mitochondria contact points and mitochondrial calcium concentrations.
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Oxidized ATP. An irreversible inhibitor of the macrophage purinergic P2Z receptor.

Marta Murgia, +4 more
- 15 Apr 1993 - 
TL;DR: In this paper, the effects of oxidized ATP on responses triggered by extracellular adenosine 5'-triphosphate (ATPe) were investigated in the mouse macrophage-like cell line J774.