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Martin S. Tallman

Researcher at Memorial Sloan Kettering Cancer Center

Publications -  948
Citations -  71451

Martin S. Tallman is an academic researcher from Memorial Sloan Kettering Cancer Center. The author has contributed to research in topics: Myeloid leukemia & Leukemia. The author has an hindex of 117, co-authored 917 publications receiving 60011 citations. Previous affiliations of Martin S. Tallman include University of Rome Tor Vergata & University of Toronto.

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Allogeneic Hematopoietic Stem Cell Transplantation Is Underutilized in Older Patients with Myelodysplastic Syndromes

TL;DR: It is highlighted that transplantation for MDS remains underutilized, particularly for candidates over the age of 65, and factors associated with a lower likelihood of undergoing transplantation included age ≥65 (P < .001), age ≥ 65 (P = .001), and <5% blasts at diagnosis (overall P = .031).
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Arsenic trioxide: its role in acute promyelocytic leukemia and potential in other hematologic malignancies.

TL;DR: Reports from China emerged showing that arsenic trioxide (ATO) was extremely effective in inducing complete remission (CR) in patients with relapsed and refractory acute promyelocytic leukemia (APL), and studies in the United States and elsewhere confirmed very high CR rates achieved with ATO.
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Pretransplantation consolidation chemotherapy decreases leukemia relapse after autologous blood and bone marrow transplants for acute myelogenous leukemia in first remission.

TL;DR: It is recommended that patients with AML in first remission receive consolidation before undergoing autotransplantation, as risks of relapse and treatment failure were lower in the patients receiving consolidation.
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Retinoic acid syndrome: a problem of the past?

TL;DR: The PETHEMA Spanish cooperative group has addressed two major questions regarding the use of retinoids in this subgroup of leukemia and focused on administering maintenance chemotherapy to all patients and minimizing chemotherapy.
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Acute myeloid leukemia presenting with panhypopituitarism or diabetes insipidus: a case series with molecular genetic analysis and review of the literature.

TL;DR: Gene sequencing of 30 genes commonly mutated in AML in three patients with available leukemia cell DNA was performed, finding one patient had no identifiable mutations, and two had RUNX1 F158S mutations.