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Martin Saxtorph Bojer

Researcher at University of Copenhagen

Publications -  37
Citations -  1336

Martin Saxtorph Bojer is an academic researcher from University of Copenhagen. The author has contributed to research in topics: Quorum sensing & Staphylococcus aureus. The author has an hindex of 18, co-authored 34 publications receiving 1052 citations. Previous affiliations of Martin Saxtorph Bojer include Statens Serum Institut & Roskilde University.

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Characterization of Klebsiella pneumoniae Type 1 Fimbriae by Detection of Phase Variation during Colonization and Infection and Impact on Virulence

TL;DR: During colonization and infection, there is pronounced selective pressure in different host environments for selection of either the type 1 fimbriated or nonfimbriate phenotype of K. pneumoniae.
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Identification of a Conserved Chromosomal Region Encoding Klebsiella pneumoniae Type 1 and Type 3 Fimbriae and Assessment of the Role of Fimbriae in Pathogenicity

TL;DR: Type 3 fimbriae were established not to be a virulence factor in uncomplicated K. pneumoniae infections and their role in development of infections in catheterized patients needs to be elucidated.
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Investigation of the putative virulence gene magA in a worldwide collection of 495 Klebsiella isolates: magA is restricted to the gene cluster of Klebsiella pneumoniae capsule serotype K1.

TL;DR: Klebsiella pneumoniae is a well-known opportunistic pathogen associated with nosocomial infections such as urinary tract infections, pneumonia and septicaemia and was reported as the most common organism isolated from pyogenic liver abscesses in two independent USA studies.
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Solonamide B inhibits quorum sensing and reduces Staphylococcus aureus mediated killing of human neutrophils.

TL;DR: In strain USA300 Solonamide B dramatically reduced the activity of α-hemolysin and the transcription of psma encoding PSMs with an 80% reduction in toxicity of supernatants towards human neutrophils and rabbit erythrocytes.
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Myricetin protects Galleria mellonella against Staphylococcus aureus infection and inhibits multiple virulence factors

TL;DR: It is reported here that myricetin, but not its glycosylated form, can remarkably decrease the production of several S. aureus virulence factors, including adhesion, biofilm formation, hemolysis and staphyloxanthin production, without interfering with growth.