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Mateus Grings

Researcher at Universidade Federal do Rio Grande do Sul

Publications -  60
Citations -  883

Mateus Grings is an academic researcher from Universidade Federal do Rio Grande do Sul. The author has contributed to research in topics: Glutathione & Oxidative stress. The author has an hindex of 15, co-authored 54 publications receiving 663 citations. Previous affiliations of Mateus Grings include University of Pittsburgh.

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Neurochemical evidence that pristanic acid impairs energy production and inhibits synaptic Na(+), K(+)-ATPase activity in brain of young rats.

TL;DR: The in vitro effects of Prist on important parameters of energy metabolism in brain cortex of young rats indicate that Prist compromises brain bioenergetics and neurotransmission.
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Glycine Intracerebroventricular Administration Disrupts Mitochondrial Energy Homeostasis in Cerebral Cortex and Striatum of Young Rats

TL;DR: The in vivo effects of intracerebroventricular administration of GLY on important parameters of energy metabolism in cerebral cortex and striatum from young rats indicate that crucial pathways for energy production and intracellular energy transfer are severely compromised by GLY.
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Neurochemical evidence that the metabolites accumulating in 3-methylcrotonyl-CoA carboxylase deficiency induce oxidative damage in cerebral cortex of young rats.

TL;DR: In vitro effects of 3MCG and 3MCA are investigated on important parameters of oxidative stress in cerebral cortex of young rats, suggesting alterations of the cellular redox homeostasis caused by the major metabolites accumulating in 3MCCD may potentially be involved in the pathophysiology of the neurological dysfunction and structural brain alterations found in patients affected by this disorder.
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Disruption of Energy Transfer and Redox Status by Sulfite in Hippocampus, Striatum, and Cerebellum of Developing Rats

TL;DR: In vitro results showed that sulfite and thiosulfate decreased CK activity, whereas sulfite also increased malondialdehyde levels in all brain structures evaluated, and impairment of energy metabolism and redox status may be important pathomechanisms involved in brain damage observed in individuals with SO deficiency.