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Matti Laine

Researcher at Åbo Akademi University

Publications -  253
Citations -  11346

Matti Laine is an academic researcher from Åbo Akademi University. The author has contributed to research in topics: Cognition & Working memory. The author has an hindex of 56, co-authored 239 publications receiving 10256 citations. Previous affiliations of Matti Laine include University of Turku & University of Helsinki.

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Music listening enhances cognitive recovery and mood after middle cerebral artery stroke

TL;DR: Recovery in the domains of verbal memory and focused attention improved significantly more in the music group than in the language and control groups, and music listening during the early post-stroke stage can enhance cognitive recovery and prevent negative mood.
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Frontal and temporal dopamine release during working memory and attention tasks in healthy humans: a positron emission tomography study using the high-affinity dopamine D2 receptor ligand [11C]FLB 457.

TL;DR: The results indicate that regionally specific components of the frontotemporal dopaminergic network are functionally involved in WM performance in humans.
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The effects of memory load on event-related EEG desynchronization and synchronization

TL;DR: The results support the assumption that the simultaneously recorded ERD/ERS responses of different narrow EEG frequency bands differ and reflect distinct aspects of information processing.
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Attention and cognitive control: Unfolding the dichotic listening story

TL;DR: A new way of defining degrees of cognitive control based on systematically varying the stimulus intensity of the right or left ear dichotic stimulus is suggested, thus parametrically varying the degree of stimulus interference and conflict when assessing the amount of Cognitive control necessary to resolve the interference.
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Y402H polymorphism of complement factor H affects binding affinity to C-reactive protein

TL;DR: In this paper, the authors examined the effects of the Y402H polymorphism on various FH functions, including reduced binding to C-reactive protein (CRP), an acute phase protein, and increased inflammation along the macular retinal pigmented epithelium-choroid interface in individuals with age-related macular degeneration.