Meizan Lai

TL;DR: A well-defined set of clinical characteristics are associated with anti-NMDA-receptor encephalitis and the pathogenesis of the disorder seems to be mediated by antibodies.

TL;DR: These studies establish the cellular mechanisms through which antibodies of patients with anti-NMDAR encephalitis cause a specific, titer-dependent, and reversible loss of NMDARs.

TL;DR: The term limbic encephalitis associated with antibodies against voltage-gated potassium channels should be changed to limbicEncephalitisassociated with LGI1 antibodies, and this disorder should be classed as an autoimmune synaptic encephalopathy.

TL;DR: GABA(B) receptor autoimmune encephalitis is a potentially treatable disorder characterised by seizures and, in some patients, associated with small-cell lung cancer and with other autoantibodies.

TL;DR: The clinical and immunological features of a novel autoantigen related to limbic encephalitis and the effect of patients' antibodies on neuronal cultures are reported.