M
Michael D. Geschwind
Researcher at University of California, San Francisco
Publications - 180
Citations - 13061
Michael D. Geschwind is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Dementia & Medicine. The author has an hindex of 46, co-authored 159 publications receiving 10415 citations. Previous affiliations of Michael D. Geschwind include University of São Paulo & University of California, Los Angeles.
Papers
More filters
Journal ArticleDOI
A clinical approach to diagnosis of autoimmune encephalitis
Francesc Graus,Maarten J. Titulaer,Ramani Balu,Susanne M. Benseler,Christian G. Bien,Tania Cellucci,Irene Cortese,Russell C. Dale,Jeffrey M. Gelfand,Michael D. Geschwind,Carol A. Glaser,Jérôme Honnorat,Romana Höftberger,Takahiro Iizuka,Sarosh R. Irani,Eric Lancaster,Frank Leypoldt,Harald Prüss,Alexander Rae-Grant,Markus Reindl,Myrna R. Rosenfeld,Kevin Rostasy,Albert Saiz,Arun Venkatesan,Angela Vincent,Klaus Peter Wandinger,Patrick Waters,J. Dalmau,J. Dalmau +28 more
TL;DR: Through logical differential diagnosis, levels of evidence for autoimmune encephalitis (possible, probable, or definite) are achieved, which can lead to prompt immunotherapy.
Journal ArticleDOI
AMPA receptor antibodies in limbic encephalitis alter synaptic receptor location
Meizan Lai,Ethan G. Hughes,Xiaoyu Peng,Lei Zhou,Amy J. Gleichman,Huidy Shu,Sabrina Matà,Daniel Kremens,Roberta Vitaliani,Michael D. Geschwind,Luis Bataller,Robert G. Kalb,Rebecca Davis,Francesc Graus,David A. Lynch,David A. Lynch,Rita J. Balice-Gordon,Josep Dalmau +17 more
TL;DR: The clinical and immunological features of a novel autoantigen related to limbic encephalitis and the effect of patients' antibodies on neuronal cultures are reported.
Journal ArticleDOI
Clinical Neurology and Epidemiology of the Major Neurodegenerative Diseases
TL;DR: This review summarizes and highlights clinical aspects of several of the most commonly encountered neurodegenerative diseases, including AD, frontotemporal dementia (FTD) and its variants, progressive supranuclear palsy (PSP), corticobasal degeneration (CBD), Parkinson's disease (PD), dementia with Lewy bodies (DLB), multiple system atrophy (MSA), and Huntington's Disease (HD).
Journal ArticleDOI
Seizures and Epileptiform Activity in the Early Stages of Alzheimer Disease
Keith A. Vossel,Alexander J. Beagle,Gil D. Rabinovici,Huidy Shu,Huidy Shu,Suzee E. Lee,Georges Naasan,Manu Hegde,Susannah Cornes,Maya L. Henry,Alexandra B. Nelson,William W. Seeley,Michael D. Geschwind,Maria Luisa Gorno-Tempini,Tina Shih,Heidi E. Kirsch,Paul A. Garcia,Bruce L. Miller,Lennart Mucke +18 more
TL;DR: Common clinical features of patients with aMCI- or AD-associated epilepsy included early age at onset of cognitive decline, early incidence of seizures in the disease course, unilateral temporal epileptic foci detected by serial/extended EEG, transient cognitive dysfunction, and good seizure control and tolerability with lamotrigine and levetiracetam.
Journal ArticleDOI
Clinicopathological correlations in corticobasal degeneration
Suzee E. Lee,Gil D. Rabinovici,Mary Catherine Mayo,Stephen M. Wilson,Stephen M. Wilson,William W. Seeley,Stephen J. DeArmond,Eric J. Huang,John Q. Trojanowski,Matthew E. Growdon,Jung Y. Jang,Manu Sidhu,Tricia See,Anna Karydas,Maria Luisa Gorno-Tempini,Adam L. Boxer,Michael W. Weiner,Michael D. Geschwind,Katherine P. Rankin,Bruce L. Miller +19 more
TL;DR: To characterize cognitive and behavioral features, physical findings, and brain atrophy patterns in pathology‐proven corticobasal degeneration (CBD) and corticOBasal syndrome (CBS) with known histopathology.