Michael G. Mason

TL;DR: The morphological phenotypes of some arr mutants suggest complex regulatory interactions and gene-specific functions among family members, suggesting a general involvement of type-A ARRs in light signal transduction.

TL;DR: Results indicate that there is functional overlap among the type-B ARRs and that they act as positive regulators of cytokinin signal transduction.

TL;DR: It is revealed that apical dominance is predominantly controlled by the shoot tip’s intense demand for sugars, which limits sugar availability to the axillary buds, and artificially increasing sucrose levels in plants represses the expression of BRANCHED1, the key transcriptional regulator responsible for maintaining bud dormancy, and results in rapid bud release.

TL;DR: Characterization of double mutants revealed differing contributions of the type B ARRs to mutant phenotypes, supporting a model in which cytokinin regulates a wide array of downstream responses through the action of a multistep phosphorelay that culminates in transcriptional regulation by ARR1, ARR10, and ARR12.

TL;DR: The evolutionarily ancient two-component system that is used by cytokinin branches to incorporate a unique family of plant-specific transcription factors is revealed.