M
Michael J. Garlepp
Researcher at Curtin University
Publications - 57
Citations - 2293
Michael J. Garlepp is an academic researcher from Curtin University. The author has contributed to research in topics: Inclusion body myositis & Human leukocyte antigen. The author has an hindex of 24, co-authored 57 publications receiving 2217 citations. Previous affiliations of Michael J. Garlepp include University of Western Australia & Royal Perth Hospital.
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Identification of differentially expressed genes in murine mesothelioma cell lines of differing tumorigenicity using suppression subtractive hybridization.
TL;DR: SSH is a powerful method for the identification of genes expressed differentially between phenotypically different tumour cell lines or clones and is shown to provide useful information in understanding genes responsible for differential tumorigenicity.
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Apolipoprotein ε alleles in sporadic inclusion body myositis: A reappraisal
Merrilee Needham,Amanda J. Hooper,Ian James,Frank M. van Bockxmeer,Frank M. van Bockxmeer,Alastair Corbett,Timothy Day,Michael J. Garlepp,Frank L. Mastaglia +8 more
TL;DR: The findings argue against a specific role for any APOEalleles in conferring susceptibility to sIBM but have demonstrated a non-significant trend towards an earlier age-of-onset in patients with the e2allele.
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Generation of an antitumour immune response to a murine mesothelioma cell line by the transfection of allogeneic mhc genes
TL;DR: The expression of allogeneic MHC molecules on a highly immunosuppressive and non‐immunogenic murine malignant mesothelioma cell line was able to stimulate rejection of tumour cells but was incapable of generating a systemic protective response against the parental cell line.
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Autoantibodies in inflammatory myopathies.
TL;DR: A variety of antibodies directed against cellular constituents, some nuclear and others cytoplasmic, have been reported in patients with inflammatory myopathies; however, it remains uncertain what role, if any, they play in the pathogenesis of these conditions.
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An analysis of the relationship between gamma delta T cell receptor V gene usage and non-major histocompatibility complex-restricted cytotoxicity.
TL;DR: A panel of five malignant mesothelioma cell lines, as well as standard tumour targets K562 and Daudi, is used to investigate some of the factors which could be involved in non‐MHC restricted cytotoxicity mediated by γδ T cells.