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Michael L. Risner

Researcher at Vanderbilt University Medical Center

Publications -  28
Citations -  356

Michael L. Risner is an academic researcher from Vanderbilt University Medical Center. The author has contributed to research in topics: Retinal ganglion cell & Medicine. The author has an hindex of 8, co-authored 19 publications receiving 240 citations. Previous affiliations of Michael L. Risner include Vanderbilt University & University of Alabama at Birmingham.

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Journal ArticleDOI

Axogenic mechanism enhances retinal ganglion cell excitability during early progression in glaucoma

TL;DR: It was found that elevations in ocular pressure caused a paradoxical increase in ganglion cell excitability, including response to light, even in cells with substantial dendritic pruning, which suggests neurodegeneration in glaucoma involves an early axogenic response that counters IOP-related stress to excitatory dendrite architecture to slow progression and maintain signaling to the brain.
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Divergent Roles of Clock Genes in Retinal and Suprachiasmatic Nucleus Circadian Oscillators

TL;DR: The retinal neural clock has a unique pattern of clock gene dependence at the tissue level that it is similar in pattern, but more severe in degree, than the SCN neural clock, with divergent clock gene regulation of rhythmic period.
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The Visual Evoked Potential is independent of surface alpha rhythm phase

TL;DR: This work calculated the VEP where stimuli were presented at four different phases of the ongoing alpha rhythm, and subtracted away the responses to null trials synchronized to the same alpha rhythm phases, creating estimates of the V EP as a function of ongoing alpha Rhythm phase.
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Protect, Repair, and Regenerate: Towards Restoring Vision in Glaucoma.

TL;DR: Recent advances in strategies that aim to restore optic nerve function and vision in glaucoma through protective, reparative, and regenerative avenues are summarized.
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Impact of Graphene on the Efficacy of Neuron Culture Substrates

TL;DR: The results demonstrate that graphene does not impede interactions between RGCs and underlying substrate matrix, such that their positive or negative effects on neuron viability and vitality are retained and graphene is a promising biosensing material for in vitro applications in neuroscience.