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Michela Gallagher

Researcher at Johns Hopkins University

Publications -  297
Citations -  33385

Michela Gallagher is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Hippocampal formation & Hippocampus. The author has an hindex of 95, co-authored 293 publications receiving 31710 citations. Previous affiliations of Michela Gallagher include University of Vermont & Texas A&M University.

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Hippocampal dependent learning ability correlates with N-methyl-D-aspartate (NMDA) receptor levels in CA3 neurons of young and aged rats.

TL;DR: It is suggested that NMDA receptor abundance in CA3 bears a critical relationship to learning mediated by the hippocampus throughout the life span, as well as the degree to which putative age‐related changes in NMDA receptors are coupled to the effects of normal aging on spatial learning.
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Hypothalamic-pituitary-adrenal axis function and corticosterone receptor expression in behaviourally characterized young and aged Long-Evans rats.

TL;DR: These data are the first to describe a coordinated decrease in GR mRNA in a functional brain system including hippocampus and related cortical areas that occurs in tandem with impairments of the HPA response to stress and cognitive decline in ageing.
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A re-examination of the role of basal forebrain cholinergic neurons in spatial working memory

TL;DR: It is indicated that selective removal of cholinergic neurons in the basal forebrain may not be sufficient to produce a deficit in spatial working memory.
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Multiple Receptors Coupled to Phospholipase C Gate Long-Term Depression in Visual Cortex

TL;DR: It is proposed that visual cortical LTD requires a minimum of PLC activity that can be supplied independently by at least three neurotransmitter systems and places PLC-linked receptors in a unique position to control the induction of LTD and provides a mechanism for gating visual cortical plasticity via extra-retinal inputs in the intact organism.
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Medial Prefrontal Cortex Is Necessary for an Appetitive Contextual Conditioned Stimulus to Promote Eating in Sated Rats

TL;DR: A critical role for vmPFC in the brain network that mediates control of conditioned motivation to eat perhaps by a mechanism akin to appetite or craving is suggested.