Michelle L. Verbsky

TL;DR: The distribution of late-flowering phenotypes in a mapping population segregating met1-1 indicates that the flowering-time phenotype is caused by the accumulation of inherited defects at loci unlinked to the met1 mutation, which led to a global reduction of cytosine methylation throughout the genome.

TL;DR: These findings are consistent with the hypotheses that the P. syringae phytotoxin coronatine acts to promote virulence by inhibiting host defense responses and by promoting lesion formation.

TL;DR: A variety of forward genetic screens have revealed predicted plant chromatin remodeling components that are involved in either differential histone acetylation or ATP-dependent SWI2/SNF2-related complexes, which have begun to provide the groundwork for determining the function of chromatin-based control in plants.

TL;DR: A screen for mutants in the signaling pathway governing resistance in Arabidopsis thaliana to the bacterial pathogen Pseudomonas syringae involved screening for suppressor mutations that restored resistance to a susceptible line carrying a mutation in the RPS2 resistance gene.

TL;DR: In this article, the authors present DNA constructs, transgenic plants containing such constructs, and methods of making the plants, which encode a polypeptide that when expressed results in the production of fatty acid compounds having anthelmintic activity.