J
Jeremy R. Haag
Researcher at Indiana University
Publications - 26
Citations - 5754
Jeremy R. Haag is an academic researcher from Indiana University. The author has contributed to research in topics: Transcription (biology) & RNA. The author has an hindex of 16, co-authored 26 publications receiving 5301 citations. Previous affiliations of Jeremy R. Haag include University of Washington & Howard Hughes Medical Institute.
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Journal ArticleDOI
Gateway-compatible vectors for plant functional genomics and proteomics.
Keith Earley,Jeremy R. Haag,Olga Pontes,Kristen E. Opper,Tom Juehne,Keming Song,Craig S. Pikaard +6 more
TL;DR: The utility of pEarleyGate destination vectors for the expression of epitope-tagged proteins that can be affinity captured or localized by immunofluorescence microscopy is demonstrated.
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Noncoding transcription by RNA polymerase Pol IVb/Pol V mediates transcriptional silencing of overlapping and adjacent genes.
TL;DR: Arabidopsis RNA polymerase IVb/Pol V, a multisubunit nuclear enzyme required for siRNA-mediated gene silencing of transposons and other repeats, transcribes intergenic and noncoding sequences, thereby facilitating heterochromatin formation andsilencing of overlapping and adjacent genes.
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Plant nuclear RNA polymerase IV mediates siRNA and DNA methylation-dependent heterochromatin formation.
Yasuyuki Onodera,Yasuyuki Onodera,Jeremy R. Haag,Thomas S. Ream,Pedro Costa Nunes,Olga Pontes,Craig S. Pikaard +6 more
TL;DR: In this article, the authors show that Pol IV helps produce siRNAs that target de novo cytosine methylation events required for facultative heter-chromatin formation and higher-order heter-romatin associations.
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Arabidopsis MET1 Cytosine Methyltransferase Mutants
Mark W. Kankel,Douglas E. Ramsey,Trevor Stokes,Susan K. Flowers,Jeremy R. Haag,Jeffrey A. Jeddeloh,Nicole C. Riddle,Michelle L. Verbsky,Eric J. Richards +8 more
TL;DR: The distribution of late-flowering phenotypes in a mapping population segregating met1-1 indicates that the flowering-time phenotype is caused by the accumulation of inherited defects at loci unlinked to the met1 mutation, which led to a global reduction of cytosine methylation throughout the genome.
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RNA polymerase V transcription guides ARGONAUTE4 to chromatin
TL;DR: The data suggest that AGO4 is guided to target loci through base-pairing of associated siRNAs with nascent Pol V transcripts, and that DEFECTIVE in MERISTEM SILENCING3, a structural maintenance of chromosomes (SMC) hinge-domain protein, functions in the assembly of Pol V transcription initiation or elongation complexes.