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Mikael Åkesson

Researcher at Swedish University of Agricultural Sciences

Publications -  73
Citations -  2934

Mikael Åkesson is an academic researcher from Swedish University of Agricultural Sciences. The author has contributed to research in topics: Population & Inbreeding. The author has an hindex of 25, co-authored 65 publications receiving 2561 citations. Previous affiliations of Mikael Åkesson include University of Edinburgh & Lund University.

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Ten years of AFLP in ecology and evolution: why so few animals?

TL;DR: A review of research areas in the study of wild species of animals where the AFLP method should be a very valuable tool to help molecular ecologists to identify when AFLP is likely to be superior to other more established methods, such as microsatellites, SNP (single nucleotide polymorphism) analyses and multigene DNA sequencing.
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Severe inbreeding depression in a wild wolf (Canis lupus) population

TL;DR: In a small, naturally restored, wild population of grey wolves in Scandinavia, founded in 1983, a pedigree for 24 of the 28 breeding pairs established in the period 1983–2002 is constructed, corresponding to 6.04 litter-size-reducing equivalents in this wolf population.
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Microsatellite diversity predicts recruitment of sibling great reed warblers

TL;DR: Evidence from wild birds is presented that genetic variation at five microsatellite loci predicts the recruitment success of siblings in great reed warblers, suggesting that the micros Satellite markers, which are generally assumed to be neutral, cosegregated with genes exhibiting genetic variation for fitness.
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Genomic consequences of intensive inbreeding in an isolated wolf population

TL;DR: Whole-genome resequencing of 97 grey wolves from a highly inbred population reveals complete homozygosity of entire chromosomes in many individuals and characterizes the genomic consequences of intensive inbreeding.
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Does linkage disequilibrium generate heterozygosity-fitness correlations in great reed warblers?

TL;DR: The present finding of a significant within‐family multilocus heterozygosity‐survival association in a nonequilibrium population supports the view that LD generates HFCs in natural populations.