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Nabil Aziz

Researcher at University of Liverpool

Publications -  43
Citations -  2567

Nabil Aziz is an academic researcher from University of Liverpool. The author has contributed to research in topics: Sperm & Male infertility. The author has an hindex of 23, co-authored 43 publications receiving 2349 citations.

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Pathogenic mechanisms in endometriosis-associated infertility.

TL;DR: The mechanisms of infertility associated with endometriosis remain controversial and include abnormal folliculogenesis, elevated oxidative stress, altered immune function, and hormonal milieu in the follicular and peritoneal environments, and reduced endometrial receptivity.
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Novel association between sperm reactive oxygen species production, sperm morphological defects, and the sperm deformity index

TL;DR: The standard semen analysis to assess sperm motility, sperm morphology, and the SDI scores is a useful tool in identifying infertile men with high seminal ROS in infertility clinics where facilities for measuring levels of seminal ROS are not available.
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Significance of sperm characteristics in the evaluation of male infertility

TL;DR: Sperm motility and concentration provide more accurate information than morphology (WHO and Tygerberg's criteria) during infertility evaluation, and redefining the reference values for concentration and morphology may significantly increase the importance of routine semen analysis.
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Critical appraisal of World Health Organization's new reference values for human semen characteristics and effect on diagnosis and treatment of subfertile men.

TL;DR: Concerns related to the new reference values for semen characteristics are discussed, including the effect on patient referral, diagnosis, and treatment of recognized conditions, such as varicocele, and on the indications for assisted reproductive technologies.
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Sperm viability, apoptosis, and intracellular reactive oxygen species levels in human spermatozoa before and after induction of oxidative stress

TL;DR: There is a differential shift of both intracellular H( 2)O(2) and O(2)(-*) in each sperm fraction that may affect sperm quality, and sperm apoptosis is related to intrusion levels, which in turn are affected by intrACEllular O(-*) levels.