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Nabil El-Sherif

Researcher at SUNY Downstate Medical Center

Publications -  257
Citations -  13915

Nabil El-Sherif is an academic researcher from SUNY Downstate Medical Center. The author has contributed to research in topics: Electrocardiography & Myocardial infarction. The author has an hindex of 60, co-authored 256 publications receiving 13148 citations. Previous affiliations of Nabil El-Sherif include Veterans Health Administration & University of Miami.

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Standards for analysis of ventricular late potentials using high-resolution or signal-averaged electrocardiography: A statement by a task force committee of the European Society of Cardiology, the American Heart Association, and the American College of Cardiology

TL;DR: No data are available about the extent to which pharmacological or nonpharmacological interventions in patients with late potentials have an impact on the incidence of sudden cardiac death, so controlled, prospective studies are required before this issue can be resolved.
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Re-entrant ventricular arrhythmias in the late myocardial infarction period. 1. Conduction characteristics in the infarction zone.

TL;DR: The study provides the first direct in vivo evidence of ventricular re-entry and demonstrates propensity for RVA and sudden death in the late myocardial infarction period.
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The electrophysiological mechanism of ventricular arrhythmias in the long QT syndrome. Tridimensional mapping of activation and recovery patterns.

TL;DR: High-resolution tridimensional isochronal mapping of both activation and repolarization patterns in puppies exposed to AP-A that developed LQTS and polymorphic ventricular tachyarrhythmias (VTs) shows that the initial beat of polymorphic VT consistently arose as focal activity from a subend cardiac site, whereas subsequent beats were due to successive subendocardial focal activity, reentrant excitation, or a combination of both.
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Characterization and Localization of Ventricular Arrhythmias Resulting from Myocardial Ischemia and Infarction

TL;DR: The ventricular arrhythmias noted 24 hours after coronary artery ligation were revealed by vagally induced atrial slowing and suppressed by rapid atrial pacing, indicating the existence of enhanced ventricular automaticity.