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Naoki Kunugita

Researcher at University of Occupational and Environmental Health Japan

Publications -  215
Citations -  5113

Naoki Kunugita is an academic researcher from University of Occupational and Environmental Health Japan. The author has contributed to research in topics: Oxidative stress & Sidestream smoke. The author has an hindex of 35, co-authored 211 publications receiving 4353 citations. Previous affiliations of Naoki Kunugita include RMIT University & University of Miyazaki.

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Carbonyl Compounds Generated from Electronic Cigarettes

TL;DR: There have been reports of many hazardous chemical compounds generated from e-cigarettes, particularly carbonyl compounds such as formaldehyde, acetaldehyde, acrolein, and glyoxal which are often found in e-cigarette aerosols.
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Heat-not-burn tobacco product use in Japan: its prevalence, predictors and perceived symptoms from exposure to secondhand heat-not-burn tobacco aerosol

TL;DR: Interest in heat-not-burn (HNB) tobacco, its prevalence, predictors of its use and symptoms from exposure to secondhand HNB tobacco aerosol in Japan are examined, finding a popular TV programme triggered IQOS diffusion in Japan.
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Determination of Carbonyl Compounds Generated from the E-cigarette Using Coupled Silica Cartridges Impregnated with Hydroquinone and 2,4-Dinitrophenylhydrazine, Followed by High-Performance Liquid Chromatography

TL;DR: It was elucidated that E-cigarettes incidentally generate high concentrations of carbonyl compounds, and the mean values were largely different from the median values.
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Bisphenol A levels in human urine.

TL;DR: An analytical high-performance liquid chromatography/fluorescence method is reported for BPA and its conjugate in human urine and on the application of this method in two student cohorts, finding that the urinary BPA levels in the students in 1992 were significantly higher than were those in 1999.
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Aldehyde dehydrogenase (ALDH) 2 associates with oxidation of methoxyacetaldehyde; in vitro analysis with liver subcellular fraction derived from human and Aldh2 gene targeting mouse.

TL;DR: In vitro MALD oxidation was examined with liver subcellular fractions from Japanese subjects who carried three different ALDH2 genotypes and Aldh2 knockout mice, which suggest that ALDH 2 is a key enzyme for Mald oxidation and ME susceptibility may be influenced by the AL DH2 genotype.