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Nathalia Bernardes

Researcher at University of São Paulo

Publications -  35
Citations -  495

Nathalia Bernardes is an academic researcher from University of São Paulo. The author has contributed to research in topics: Blood pressure & Aerobic exercise. The author has an hindex of 11, co-authored 29 publications receiving 402 citations. Previous affiliations of Nathalia Bernardes include University of Manitoba & Universidade São Judas Tadeu.

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Insulin resistance: an additional risk factor in the pathogenesis of cardiovascular disease in type 2 diabetes.

TL;DR: It evidently becomes important to unravel the mechanisms of the association between IR and CVDs in order to formulate novel efficient drugs to treat patients suffering from insulin resistance-mediated cardiovascular diseases.
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Parasympathetic dysfunction is associated with insulin resistance in fructose-fed female rats

TL;DR: Data provided new information regarding the role of parasympathetic dysfunction associated with insulin resistance in the development of early metabolic and cardiovascular alterations induced by a high fructose diet.
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Positive effect of combined exercise training in a model of metabolic syndrome and menopause: autonomic, inflammatory, and oxidative stress evaluations.

TL;DR: In conclusion, the association of risk factors promoted an additional impairment in metabolic, cardiovascular, autonomic, inflammatory, and oxidative stress parameters and combined exercise training was able to attenuate these dysfunctions.
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Resistance or aerobic training decreases blood pressure and improves cardiovascular autonomic control and oxidative stress in hypertensive menopausal rats

TL;DR: Both RT and AT can decrease arterial pressure in a model of hypertension and menopause; although, at different magnitudes this decrease was related to attenuated autonomic dysfunction in association with cardiac oxidative stress improvement in both exercise protocols.
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Cardiovascular autonomic dysfunction and oxidative stress induced by fructose overload in an experimental model of hypertension and menopause

TL;DR: Fructose overload promoted an impairment in cardiac autonomic modulation associated with inflammation and oxidative stress in hypertensive rats undergoing ovarian hormone deprivation in an experimental model of hypertension and menopause.