O
Ole Hamberg
Researcher at University of Copenhagen
Publications - 14
Citations - 2187
Ole Hamberg is an academic researcher from University of Copenhagen. The author has contributed to research in topics: Urea & Liver function. The author has an hindex of 8, co-authored 13 publications receiving 1783 citations.
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Nutritional risk screening (NRS 2002): a new method based on an analysis of controlled clinical trials.
TL;DR: The screening system appears to be able to distinguish between trials with a positive effect vs no effect, and it can therefore probably also identify patients who are likely to benefit from nutritional support.
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Effects of an increase in protein intake on hepatic efficacy for urea synthesis in healthy subjects and in patients with cirrhosis.
TL;DR: The study shows that an increase in protein intake selectively increases liver function with regard to disposal of amino nitrogen; the mechanism is qualitatively intact but quantitatively deficient in patients with cirrhosis of the liver, and does not seem to depend on glucagon.
Journal ArticleDOI
Effects of insulin and glucose on urea synthesis in normal man, independent of pancreatic hormone secretion
Ole Hamberg,Hendrik Vilstrup +1 more
TL;DR: The inhibitory effect of insulin and glucose on hepatic amino- to urea-nitrogen conversion independent of endogenous insulin and glucagon secretion means that the hepatic component of the amino-N-sparing effect of glucose depends on hyperglycaemia but not on hyperinsulinaemia.
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Effects of budesonide and prednisolone on hepatic kinetics for urea synthesis.
TL;DR: Prednisolone administration led to increased levels of amino acids in blood and loss of N as urea, the latter in part due to a specific hepatic mechanism as shown by the increased FHNC, making treatment with budesonide superior to that of conventional glucocorticoids in this respect.
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Increased hepatic urea synthesis in patients with active inflammatory bowel disease.
TL;DR: The results show that the liver function related to conversion of amino-nitrogen to urea is increased in patients with active inflammatory bowel disease, and the accelerated hepatic amino- Nitrogen conversion contributes to the less efficient nitrogen economy in Patients with activeinflammatory bowel disease.