O
Ourania M. Andrisani
Researcher at Purdue University
Publications - 82
Citations - 4183
Ourania M. Andrisani is an academic researcher from Purdue University. The author has contributed to research in topics: CREB & Hepatitis B virus. The author has an hindex of 38, co-authored 77 publications receiving 3782 citations. Previous affiliations of Ourania M. Andrisani include Stanford University & Vanderbilt University.
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Vitamin A Metabolites Induce Gut-Homing FoxP3+ Regulatory T Cells
TL;DR: Results identify retinoids as positive regulatory factors for generation of gut-homing FoxP3+ T cells and a unique cellular feature of these regulatory T cells is their high expression of Gut-Homing receptors that are important for migration to the mucosal tissues particularly the small intestine.
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A secondary phosphorylation of CREB341 at Ser129 is required for the cAMP-mediated control of gene expression. A role for glycogen synthase kinase-3 in the control of gene expression.
TL;DR: It is proposed that the hierarchical phosphorylation at the PKA and GSK-3 sites of CREB are essential for cAMP control ofCREB.
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E1A 13S and 12S mRNA products made in Escherichia coli both function as nucleus-localized transcription activators but do not directly bind DNA.
Blair Ferguson,Bernd Krippl,Ourania M. Andrisani,Nicholas C. Jones,Heiner Westphal,Martin Rosenberg +5 more
TL;DR: Although both E1A gene products localized to the nucleus and stimulated adenovirus gene transcription, these proteins did not directly bind to DNA under conditions in which a known DNA-binding protein, the human c-myc gene product, bound DNA efficiently.
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The hepatitis B virus X protein targets the basic region-leucine zipper domain of CREB.
TL;DR: The experiments demonstrate that pX titers the way CREB interacts with the CRE DNA and suggest that the basic, DNA-binding region of CREB is the target of pX, and support the significance of the CREB-pX protein-protein interactions in vivo.
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The transcriptional function of the hepatitis B virus X protein and its role in hepatocarcinogenesis (Review).
TL;DR: The transactivation function of pX is described and its role in hepatocarcinogenesis is described, which is regarded as a promiscuous transactivator, acting via a dual mechanism.