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Pauline M Emmett

Researcher at University of Bristol

Publications -  272
Citations -  21854

Pauline M Emmett is an academic researcher from University of Bristol. The author has contributed to research in topics: Pregnancy & Breast feeding. The author has an hindex of 75, co-authored 261 publications receiving 20210 citations. Previous affiliations of Pauline M Emmett include Medical Research Council & University of Texas Southwestern Medical Center.

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Early life risk factors for obesity in childhood: cohort study

TL;DR: Eight factors in early life are associated with an increased risk of obesity in childhood, including parental obesity and catch-up growth.
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Association between postnatal catch-up growth and obesity in childhood: prospective cohort study.

TL;DR: In this contemporary well nourished cohort, catch-up growth was predicted by factors relating to intrauterine restraint of fetal growth, and children who showed catch- up growth between zero and two years were fatter and had more central fat distribution at five years than other children.
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Maternal seafood consumption in pregnancy and neurodevelopmental outcomes in childhood (ALSPAC study): an observational cohort study.

TL;DR: Risks from the loss of nutrients were greater than the risks of harm from exposure to trace contaminants in 340 g seafood eaten weekly, suggesting that advice to limit seafood consumption could actually be detrimental.
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Effect of inadequate iodine status in UK pregnant women on cognitive outcomes in their children: results from the Avon Longitudinal Study of Parents and Children (ALSPAC)

TL;DR: The results show the importance of adequate iodine status during early gestation and emphasise the risk that iodine deficiency can pose to the developing infant, even in a country classified as only mildly iodine deficient.
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Size at Birth and Early Childhood Growth in Relation to Maternal Smoking, Parity and Infant Breast-Feeding: Longitudinal Birth Cohort Study and Analysis

TL;DR: Early postnatal growth rates are strongly influenced by a drive to compensate for antenatal restraint or enhancement of fetal growth by maternal-uterine factors, particularly in breast- and bottle-fed infants.