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Pavel M. Balaban

Researcher at Russian Academy of Sciences

Publications -  234
Citations -  2553

Pavel M. Balaban is an academic researcher from Russian Academy of Sciences. The author has contributed to research in topics: Helix lucorum & Synaptic plasticity. The author has an hindex of 25, co-authored 224 publications receiving 2266 citations. Previous affiliations of Pavel M. Balaban include AT&T Labs & Saint Petersburg State Polytechnic University.

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Journal Article

[Fast and aimed delivery of voltage-sensitive dyes to mammalian brain slices by biolistic techniques].

TL;DR: A new protocol based on rapid biolistic delivery of VSDs is reported, which is optimal for further recordings of optical signals from live neurons of rat brain slices and can be used for rapid local delivery of any lipophilic and water-insoluble substances into live cells, further optical recording of neural activity, and analysis of potential propagation in a nerve cell.
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Sensitization and habituation in command neurons of the defensive reflex in Helix lucorum.

TL;DR: The cause of sensitization of the command neurons is long-lasting depolarization in response to tactile stimulation, arising after the spike discharge of the cell and facilitating responses to subsequent stimulation.
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Experiments with Snails Add to Our Knowledge about the Role of aPKC Subfamily Kinases in Learning

TL;DR: The sequence of mRNA encoding PKMζ homolog in land snail Helix lucorum is established, and it is discovered that this mRNA has at least two isoforms, and one of them lacks sequence encoding C1 domain.
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Selective blockade of gene expression in a single identified snail neuron

TL;DR: High stability of the morpholino oligos in the cell represents a highly efficient tool for a specific long-term blockade of gene expression in molluscan neurons, suggesting significant selective blockade of production of the HCS2-encoded peptide.
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Serotonin-induced changes in the action of functionally distinct neurons in Helix pomatia

Pavel M. Balaban
- 01 Jan 1988 - 
TL;DR: The duration of action potentials in sensory neurons was found to increase under the effects of serotonin (and this could underly the rise in EPSP amplitude), although that of interneuron and motoneuron spikes did not change.