P
Pavel M. Balaban
Researcher at Russian Academy of Sciences
Publications - 234
Citations - 2553
Pavel M. Balaban is an academic researcher from Russian Academy of Sciences. The author has contributed to research in topics: Helix lucorum & Synaptic plasticity. The author has an hindex of 25, co-authored 224 publications receiving 2266 citations. Previous affiliations of Pavel M. Balaban include AT&T Labs & Saint Petersburg State Polytechnic University.
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[Low-threshold area of action potential generation in the somatic membrane of mollusk neurons].
TL;DR: Heterogeneity of the somatic membrane of subesophageal ganglionic neurons of Helix lucorum was studied by extracellular electrical stimulation and the threshold of action potential generation was shown to depend on the direction of the extrace cellular current.
Journal ArticleDOI
Decrease of effectivity of "competing" synaptic input requires protein synthesis.
Pavel M. Balaban,Maksimova Oa +1 more
TL;DR: The results suggest that the heterosynaptically evoked decrease of synaptic response amplitude may be due to involvement of postsynaptic protein synthesis-dependent mechanisms.
Posted ContentDOI
Central Targeting of Channelrodopsin2 by Motif of Potassium Channel Kv2.1 Can Be Altered Due to Overexpression of Construct
TL;DR: It is suggested that observed mislocalization was caused by overexpression of the construct, and the possibility of “incorrect” targeting of CHR2 by the potassium channel motif Kv2.1 should be taken into account when using this construct in optogenetic experiments.
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Optogenetic Stimulation of the Axons of Visual Cortex and Hippocampus Pyramidal Neurons in Living Brain Slices
TL;DR: The use of local activation of axons using an optogenetic stimulation method to stimulate the axons of pyramidal neurons in the central nervous system of mammals is reported here.
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Amyloid Aβ25-35 Aggregates Say ‘NO’ to Long-Term Potentiation in the Hippocampus through Activation of Stress-Induced Phosphatase 1 and Mitochondrial Na+/Ca2+ Exchanger
TL;DR: H hippocampal NO production could be another marker for the impairment of synaptic plasticity in amyloidosis-related states, and kinase–phosphatase balance management could be a promising strategy for the compensation of Aβ25-35-driven deteriorations.