P
Per Venge
Researcher at Uppsala University
Publications - 457
Citations - 34544
Per Venge is an academic researcher from Uppsala University. The author has contributed to research in topics: Eosinophil cationic protein & Eosinophil. The author has an hindex of 86, co-authored 454 publications receiving 33109 citations. Previous affiliations of Per Venge include University of Helsinki & Karolinska Institutet.
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Journal ArticleDOI
Gluten sensitivity in patients with IgA nephropathy
Hilde Kloster Smerud,Bengt Fellström,Roger Hällgren,Sonia Osagie,Per Venge,Gudjon Kristjánsson +5 more
TL;DR: It is concluded that approximately one-third of IgAN patients have a rectal mucosal sensitivity to gluten, but without signs of coeliac disease, and it is hypothesize that such sub-clinical inflammation to gluten might be involved in the pathogenesis of IgAn in a subgroup of patients.
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Factors Influencing the 99th Percentile of Cardiac Troponin I Evaluated in Community-Dwelling Individuals at 70 and 75 Years of Age
TL;DR: Among elderly community dwellers, higher concentrations were seen in men and individuals with prevalent CVD, and aging contributes to increasing concentrations, given the pronounced changes seen with increasing age across all subgroups.
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Serum eosinophil cationic protein in relation to bronchial asthma in a young Swedish population
Eythor Björnsson,Christer Janson,Christer Janson,Lena Håkansson,Lena Håkansson,I. Enander,Per Venge,Per Venge,Gunnar Boman,Gunnar Boman +9 more
TL;DR: Both B‐Eos and S‐ECP and their interrelationship may be of value in assessing the activity of asthma, however, their role in disease management was not established in this cross‐sectional study.
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Granulocyte colony-stimulating factor (G-CSF) induces the production of cytokines in vivo.
TL;DR: It is suggested that G‐CSF induces the production of TNF‐α and GM‐CSf in vivo, and that this production may be due to the co‐effects of endothelial cells and whole blood under the influence of G‐ CSF through an as yet unknown network of cells and cytokines.
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Relationship between sleep-disordered breathing and markers of systemic inflammation in women from the general population.
TL;DR: The results indicate that intermittent hypoxia, and not the AHI, is related to systemic inflammation seen in OSAS, and for women from the general population there is an independent correlation between SDB and inflammation, even after adjusting for obesity.