P
Per Venge
Researcher at Uppsala University
Publications - 457
Citations - 34544
Per Venge is an academic researcher from Uppsala University. The author has contributed to research in topics: Eosinophil cationic protein & Eosinophil. The author has an hindex of 86, co-authored 454 publications receiving 33109 citations. Previous affiliations of Per Venge include University of Helsinki & Karolinska Institutet.
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Journal ArticleDOI
Myeloperoxidase in human lung lavage. I. A marker of local neutrophil activity.
TL;DR: Strong evidence suggests that MPO in BAL originates from lung neutrophils and that BAL MPO content may be used to estimate the neutrophil presence or activation in epithelium lining fluid.
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Eosinophil peroxidase produces hypobromous acid in the airways of stable asthmatics
Ruth E. Aldridge,Timothy Chan,Christine J. van Dalen,Revathy Senthilmohan,Marti Winn,Per Venge,G. Ian Town,Anthony J. Kettle +7 more
TL;DR: It is concluded that eosinophil peroxidase produces substantial amounts of hypobromous acid in the airways of stable asthmatics, and this highly reactive oxidant is a strong candidate for exacerbating inflammatory tissue damage in the lung.
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The extracellular deposition of mast cell products is increased in hypertrophic airways smooth muscles in allergic asthma but not in nonallergic asthma
TL;DR: The aim of this investigation was to study the distribution of mast cells in different compartments in the bronchial mucosa of allergic and nonallergic asthma in relation to airways remodeling.
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Indoor air pollutants in schools: nasal patency and biomarkers in nasal lavage.
TL;DR: There is growing concern about the respiratory health aspects of the indoor air quality in schools and the need to take action to protect students' respiratory health.
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Degranulation of eosinophils from pollen-atopic patients with asthma is increased during pollen season
TL;DR: Eosinophils and, to some extent, neutrophils from birch pollen-atopic subjects have an increased propensity to secrete their granule proteins during a pollen season, and it is suggested that these cells have been primed as a consequence of allergen exposure.