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Per Venge

Researcher at Uppsala University

Publications -  457
Citations -  34544

Per Venge is an academic researcher from Uppsala University. The author has contributed to research in topics: Eosinophil cationic protein & Eosinophil. The author has an hindex of 86, co-authored 454 publications receiving 33109 citations. Previous affiliations of Per Venge include University of Helsinki & Karolinska Institutet.

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Blood eosinophils and eosinophil cationic protein after acute myocardial infarction or corticosteroid administration.

TL;DR: The results suggest that ECP is released from eosinophils in vivo, and the raised levels of S‐ECP in patients with acute myocardial infarction are probably due to the active participation of eosInophil in the inflammatory process.
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Bronchial mucosal mast cells in asymptomatic smokers relation to structure, lung function and emphysema

TL;DR: Smoking is associated with an increase of mast cells in all compartments of the bronchial mucosa, including smooth muscle, and this is related to altered airway structure and function.
Journal Article

Eosinophil involvement in rheumatoid arthritis as reflected by elevated serum levels of eosinophil cationic protein.

TL;DR: Although eosinophils are not a notable feature of the synovial membrane infiltrate or cellular joint exudate, data obtained indirectly indicates their participation in the inflammatory reaction in RA.
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Interleukin-17A mRNA and protein expression within cells from the human bronchoalveolar space after exposure to organic dust.

TL;DR: Exposure to organic dust increases local IL-17A mRNA and because there is intracellular expression in BAL lymphocytes, this suggests that IL- 17A protein can originate from lymphocytes within the human bronchoalveolar space through its neutrophil-accumulating effect.
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Troponin-Specific Autoantibody Interference in Different Cardiac Troponin I Assay Configurations

TL;DR: A novel cTnI assay deviating from the conventional IFCC-recommended midfragment approach substantially improves cTNI detection in samples containing cTtnAAb and confirms the reduced concentrations expected from the low analytical recoveries.