P
Per Venge
Researcher at Uppsala University
Publications - 457
Citations - 34544
Per Venge is an academic researcher from Uppsala University. The author has contributed to research in topics: Eosinophil cationic protein & Eosinophil. The author has an hindex of 86, co-authored 454 publications receiving 33109 citations. Previous affiliations of Per Venge include University of Helsinki & Karolinska Institutet.
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Journal ArticleDOI
Circulating Cytokines and Granulocyte-Derived Enzymes During Complex Heart Surgery: A Clinical Study with Special Reference to Heparin-Coating of Cardiopulmonary Bypass Circuits
TL;DR: Significantly increased inflammatory response was found during complex heart operations even with use of heparin-coated CPB sets, and the heparIn-coating of circuits seems to diminish cytokine production.
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Admission Troponin T and measurement of ST-segment resolution at 60 min improve early risk stratification in ST-elevation myocardial infarction
Erik Björklund,Bertil Lindahl,Per Johanson,Tomas Jernberg,Ann-Marie Svensson,Per Venge,Lars Wallentin,Mikael Dellborg +7 more
TL;DR: Both tnT on admission and ST-segment resolution after 60min are strong predictors of one-year mortality and the combination of both gives additive early information about prognosis and further improves risk stratification.
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Gut mucosal granulocyte activation precedes nitric oxide production: studies in coeliac patients challenged with gluten and corn.
TL;DR: Mucosal activation of neutrophils and eosinophils precedes pronounced enhancement of mucosal NO production after rectal wheat gluten challenge in patients with coeliac disease.
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Effects of heparin coating on the expression of CD11b, CD11c and CD62L by leucocytes in extracorporeal circulation in vitro
TL;DR: In this in vitro ECC model, heparin coating of oxygenator/tubing sets reduced leucocytes activation and leucocyte adhesion-related phenomena, suggesting interacting regulatory pathways in the process of neutrophil adhesion, activation and degranulation.
Journal Article
Blood eosinophil leucocyte and eosinophil cationic protein. In vivo study of the influence of beta-2-adrenergic drugs and steroid medication.
Ronald Dahl,Per Venge +1 more
TL;DR: Beta-2-adrenergic drugs, salbutamol and terbutaline, had an eosinopenic effect and were able to decrease S-ECP concentration, indicating that only the adrenergic stimulators affect leucocyte secretion.