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Peter J. Coote

Researcher at University of St Andrews

Publications -  61
Citations -  6922

Peter J. Coote is an academic researcher from University of St Andrews. The author has contributed to research in topics: Antibiotics & Lysostaphin. The author has an hindex of 34, co-authored 60 publications receiving 6431 citations. Previous affiliations of Peter J. Coote include University of Bedfordshire.

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A study of the minimum inhibitory concentration and mode of action of oregano essential oil, thymol and carvacrol.

TL;DR: The minimum inhibitory concentration of oregano essential oil and two of its principle components, i.e. thymol and carvacrol, against Pseudomonas aeruginosa and Staphylococcus aureus was assessed by using an innovative technique.
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Preservative agents in foods. Mode of action and microbial resistance mechanisms.

TL;DR: The mode of action of both chemical and biological (nature-derived) preservatives and the stress response mechanisms induced by these compounds in microorganisms of concern to the food industry are discussed.
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The Pdr12 ABC transporter is required for the development of weak organic acid resistance in yeast

TL;DR: This is the first demonstration that regulated expression of a eukaryotic ABC transporter mediates weak organic acid resistance development, the cause of widespread food spoilage by yeasts, and the data suggest that the inhibition of this transporter could be a strategy for preventingFood spoilage.
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Activity of the plasma membrane H(+)-ATPase and optimal glycolytic flux are required for rapid adaptation and growth of Saccharomyces cerevisiae in the presence of the weak-acid preservative sorbic acid.

TL;DR: Data are consistent with the idea that adaptation by yeast cells to sorbic acid is dependent on the restoration of internal pH via the export of protons by the membrane H+-ATPase in an energy-demanding process and the generation of sufficient ATP to drive this process and still allow growth.
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Acid tolerance in Listeria monocytogenes: the adaptive acid tolerance response (ATR) and growth-phase-dependent acid resistance

TL;DR: Listeria monocytogenes acquired increased acid tolerance during exponential growth upon exposure to sublethal acid stress, a response designated the acid tolerance response (ATR), and developed increased acid resistance upon entry into the stationary phase; this response appeared to be independent of the pH-dependent ATR seen during exponential Growth.