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Peter J.E.H.M. Kitslaar

Researcher at Maastricht University

Publications -  61
Citations -  4503

Peter J.E.H.M. Kitslaar is an academic researcher from Maastricht University. The author has contributed to research in topics: Duplex scanning & Intimal hyperplasia. The author has an hindex of 32, co-authored 61 publications receiving 4323 citations. Previous affiliations of Peter J.E.H.M. Kitslaar include Maastricht University Medical Centre.

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Accumulation of Ultrasmall Superparamagnetic Particles of Iron Oxide in Human Atherosclerotic Plaques Can Be Detected by In Vivo Magnetic Resonance Imaging

TL;DR: Accumulation of USPIOs in macrophages in predominantly ruptured and rupture-prone human atherosclerotic lesions caused signal decreases in the in vivo MR images.
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Differential Expression of Bone Matrix Regulatory Proteins in Human Atherosclerotic Plaques

TL;DR: The observed expression patterns suggest a tight regulation of the expression of bone matrix regulatory proteins during human atherogenesis, which might suggest a regulatory role of these proteins not only in osteoclastogenesis but also in atherosclerotic calcification.
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Intimal Hyperplasia in Vascular Grafts

TL;DR: Intimal hyperplasia is now recognised as the main cause of thrombotic complications occurring between 2 and 24 months after compliance mismatch, and some solutions have had promising results with respect to improved pata vascular intervention.
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Serum C-Reactive Protein Level Is Associated With Abdominal Aortic Aneurysm Size and May Be Produced by Aneurysmal Tissue

TL;DR: This is the first report showing that serum hs CRP is associated with aneurysmal size and that—in at least some patients—CRP may be produced by aneurYSmal tissue, suggesting that serum CRP may serve as a marker of AAA disease and that CRP produced in vascular tissue might contribute to aneurym formation.
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Biphasic pattern of cell turnover characterizes the progression from fatty streaks to ruptured human atherosclerotic plaques.

TL;DR: In stable atherosclerotic lesions, DNA synthesis is an early event, while apoptosis is a late event, and Ruptured plaques show a second peak of cell turnover.