P
Piotr Głuszko
Researcher at Jagiellonian University
Publications - 80
Citations - 1978
Piotr Głuszko is an academic researcher from Jagiellonian University. The author has contributed to research in topics: Rheumatoid arthritis & Ankylosing spondylitis. The author has an hindex of 18, co-authored 72 publications receiving 1756 citations. Previous affiliations of Piotr Głuszko include Temple University & University of Pennsylvania.
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Practical guidelines for the supplementation of vitamin D and the treatment of deficits in Central Europe - recommended vitamin D intakes in the general population and groups at risk of vitamin D deficiency
Paweł Płudowski,Elzbieta Karczmarewicz,Milan Bayer,Graham D. Carter,Danuta Chlebna-Sokół,Justyna Czech-Kowalska,Romuald Dębski,Tamás Decsi,Anna Dobrzańska,Edward Franek,Piotr Głuszko,William B. Grant,Michael F. Holick,Liudmila Yankovskaya,Jerzy Konstantynowicz,Janusz Książyk,Krystyna Księżopolska-Orłowska,Andrzej Lewiński,Mieczysław Litwin,Szimonetta Lohner,Roman S. Lorenc,Jacek Łukaszkiewicz,Ewa Marcinowska-Suchowierska,Andrzej Milewicz,Waldemar Misiorowski,Michał Nowicki,Vladyslav Povoroznyuk,Piotr Rozentryt,Ema Rudenka,Yehuda Shoenfeld,Piotr Socha,Bogdan Solnica,Mieczysław Szalecki,Marek Tałałaj,Szabolcs Várbíró,Michał A. Żmijewski +35 more
TL;DR: Wright et al. as mentioned in this paper show that wiele korzyści wynikających z dzialania witaminy D na organizm czlowieka na wszystkichetapach jego zycia wiekszośc badan epidemiologicznych sugeruje, ze niedobor witaminą D, ktore przeslano do czlonkow Komitetu Naukowego konfere
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Successful therapy of advanced arteriosclerosis obliterans with prostacyclin
Andrzej Szczeklik,S. Skawinski,Piotr Głuszko,R. Nizankowski,J. Szczeklik,Ryszard J. Gryglewski +5 more
TL;DR: Five patients with advanced arteriosclerosis obliterans of the lower extremities, as evidenced by resting pain, ischaemic ulcers, and focal necrosis, received intra-arterial infusions of prostacyclin at doses of 5--10 ng/kg/min for 72 h, and pain at rest had disappeared in all patients within 2 days of termination of the infusion.
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Aprotinin inhibits the contact, neutrophil, and platelet activation systems during simulated extracorporeal perfusion.
Y. T. Wachtfogel,Umberto Kucich,C. E. Hack,Piotr Głuszko,S Niewiarowski,Robert W. Colman,L. H. Edmunds,B. P. Bidstrup,R. H. Bartlett,Safuh Attar +9 more
TL;DR: Aprotinin in high doses completely inhibited kallikrein-CĪ-inhibitor complex activation of neutrophils and partially inhibited complement-induced activation, thus attenuating the "whole body inflammatory response" associated with cardiopulmonary bypass.
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Human neutrophil degranulation during extracorporeal circulation
Y. T. Wachtfogel,Y. T. Wachtfogel,Umberto Kucich,Umberto Kucich,J Greenplate,J Greenplate,Piotr Głuszko,Piotr Głuszko,William R. Abrams,William R. Abrams,George Weinbaum,George Weinbaum,R. K. Wenger,R. K. Wenger,B. Rucinski,B. Rucinski,S Niewiarowski,S Niewiarowski,L. H. Edmunds,L. H. Edmunds +19 more
TL;DR: Inhibition in the simulated circuit by prostaglandin E1 (PGE1) and lidocaine suggests different mechanisms for release of neutrophil-specific proteins, which are explored during simulated in vitro extracorporeal circulation and during clinical cardiopulmonary bypass.
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Fibrinogen receptors in platelet adhesion to surfaces of extracorporeal circuit.
Piotr Głuszko,B. Rucinski,J Musial,R. K. Wenger,Alvin H. Schmaier,Robert W. Colman,L. H. Edmunds,Stefan Niewiarowski +7 more
TL;DR: Exposure of fibrinogen receptors associated with GPIIb-GPIIIa complex contributes to platelet consumption during cardiopulmonary bypass, and beta-Thromboglobulin antigen (beta TG) loss from platelets was associated with thrombocytopenia.