R
Raffaele Bruno
Researcher at University of Pavia
Publications - 322
Citations - 14690
Raffaele Bruno is an academic researcher from University of Pavia. The author has contributed to research in topics: Medicine & Hepatitis C. The author has an hindex of 52, co-authored 275 publications receiving 11262 citations. Previous affiliations of Raffaele Bruno include University of Modena and Reggio Emilia.
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Journal ArticleDOI
The pavia consensus statement.
Paul A. Volberding,Robert L. Murphy,Giuseppe Barbaro,Giorgio Barbarini,Raffaele Bruno,Augusto Cirelli,Peter F. Currie,Gabriella Di Lorenzo,Massimo Fantoni,Gaetano Filice,Massimo Galli,Benvenuto Grisorio,Mauro Moroni,Recusani F,Paolo Sacchi,D. Scevola,James H. Stein,Donato Torre,Daniel Vittecoq +18 more
TL;DR: The intersection of cardiovascular disease and HIV infection is common and complex, yet inadequately understood, and the bilateral nature of these interactions is an important issue considered in the present report.
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Emergency Department and Out-of-Hospital Emergency System (112-AREU 118) integrated response to Coronavirus Disease 2019 in a Northern Italy centre.
Stefano Perlini,Fabrizio Canevari,Sergio Cortesi,Vito Sgromo,Antonella Brancaglione,Enrico Contri,Pietro Pettenazza,Francesco Salinaro,Francesco Speciale,Giuseppe Maria Sechi,Claudio Mare,Sara Cutti,Viola Novelli,Carlo Marena,Alba Muzzi,Raffaele Bruno,Alessandra Palo +16 more
TL;DR: The Pavia Emergency Medicine network should be ready to finely tune their usual cooperation to respond to a sudden and overwhelming increase in the healthcare needs brought about by a pandemia like the current one and should shape and reinforce the future.
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COVID-19: what if the brain had a role in causing the deaths?
TL;DR: In the last few weeks Italy first, and then several other countries across the world, have been swept up by the deadly wave of the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), which causes the illness named COVID-19, from the acronym CO (corona) VI (virus) D (disease) and 19 (year of the virus identification).
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SARS-CoV-2 specific T-cell immunity in COVID-19 convalescent patients and unexposed controls measured by ex vivo ELISpot assay.
Irene Cassaniti,Elena Percivalle,Federica Bergami,Antonio Piralla,Giuditta Comolli,Raffaele Bruno,Marco Vecchia,Margherita Sambo,Marta Colaneri,Valentina Zuccaro,Marco Benazzo,Carlo Robotti,Anna Calastri,Eugenia Maiorano,Alessandro Ferrari,Giuseppe Cambiè,Fausto Baldanti +16 more
TL;DR: Cross-reactive SARS-CoV-2 T-cell response in uninfected patients may be due to previous infections with other common coronaviruses, and the data suggest that long-term SAR’sCOVID-19 infection might accompany a waning humoral response.
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Beyond the gut bacterial microbiota: The gut virome
P. Columpsi,Paolo Sacchi,Valentina Zuccaro,Serena Cima,Cristina Sarda,M. Mariani,Andrea Gori,Raffaele Bruno +7 more
TL;DR: The gastrointestinal tract is colonized with a highly different population of bacterial, viral, ad fungal species; viruses are reported to be dominant and phages could elicit innate antiviral immune responses.