R
Ramesh Chandra
Researcher at University of Delhi
Publications - 734
Citations - 19255
Ramesh Chandra is an academic researcher from University of Delhi. The author has contributed to research in topics: Chemistry & Medicine. The author has an hindex of 66, co-authored 620 publications receiving 16293 citations. Previous affiliations of Ramesh Chandra include Central Drug Research Institute & Stanford University.
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Damping studies in fiber-reinforced composites : a review
TL;DR: The status of research on damping in fiber-reinforced composite materials and structures with emphasis on polymer composites has been reviewed in this article, where the authors present damping studies involving macromechanical, micro-mechanical and viscoelastic (relaxation and creep) approaches.
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Optimizing the migration of virtual computers
TL;DR: This paper shows how to quickly move the state of a running computer across a network, including the state in its disks, memory, CPU registers, and I/O devices, and calls this state a capsule, and suggests that efficient capsule migration can improve user mobility and system management.
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Induced pluripotent stem cells: applications in regenerative medicine, disease modeling, and drug discovery
TL;DR: The applications of iPSCs in regenerative medicine, disease modeling, and drug discovery are enormous and should be explored in a more comprehensive manner.
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Biodegradation of maleated linear low-density polyethylene and starch blends
Ramesh Chandra,Renu Rustgi +1 more
TL;DR: In this paper, a modified linear low-density polyethylene (LLDPE) and a mixture of corn starch in varying concentrations (between 10 and 60%) was blended with maleic anhydride (MA) in a torque rheometer.
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Nucleus Accumbens Medium Spiny Neuron Subtypes Mediate Depression-Related Outcomes to Social Defeat Stress
T. Chase Francis,Ramesh Chandra,Danielle M. Friend,Eric Finkel,Genesis Dayrit,Genesis Dayrit,Jorge Miranda,Jorge Miranda,Julie M. Brooks,Sergio D. Iñiguez,Patricio O'Donnell,Alexxai V. Kravitz,Mary Kay Lobo +12 more
TL;DR: It is demonstrated that the frequency of excitatory synaptic input is decreased in D1-MSNs and increased in D2-MSNS in mice displaying depression-like behaviors after CSDS, and bidirectional alteration of D 1-MSN activity promotes opposite behavioral outcomes to chronic social stress.