scispace - formally typeset
R

Reinhard Bauer

Researcher at University of Jena

Publications -  234
Citations -  6001

Reinhard Bauer is an academic researcher from University of Jena. The author has contributed to research in topics: Cerebral blood flow & Innexin. The author has an hindex of 40, co-authored 228 publications receiving 5435 citations. Previous affiliations of Reinhard Bauer include University of Texas MD Anderson Cancer Center & University of Regensburg.

Papers
More filters
Journal ArticleDOI

Adaptive AR modeling of nonstationary time series by means of Kalman filtering

TL;DR: An adaptive on-line procedure is presented for autoregressive (AR) modeling of nonstationary multivariate time series by means of Kalman filtering and an application with experimental EEG data supported observations that the development of coherences among cell assemblies of the brain is a basic element of associative learning or conditioning.
Journal ArticleDOI

Cloning and characterization of a second AP-2 transcription factor: AP-2 beta

TL;DR: Analysis of embryonic and adult mice demonstrate that two different AP-2 transcription factors are specifically expressed during differentiation of many neural, epidermal and urogenital tissues.
Journal ArticleDOI

Transporters MCT8 and OATP1C1 maintain murine brain thyroid hormone homeostasis

TL;DR: Mct8/Oatp1c1 DKO mice characterized mice deficient for both TH transporters, MCT8 and OATP1C1, provide a basis to study the pathogenic mechanisms underlying AHDS and underscore the importance of THtransporters for proper brain development
Journal ArticleDOI

Comparison of linear signal processing techniques to infer directed interactions in multivariate neural systems

TL;DR: The performances of four representative multivariate linear signal processing techniques in the time and frequency domain have been investigated and both methods have been shown to be most suitable for this application to brain neural networks based on the model systems investigated.
Journal ArticleDOI

An alternatively spliced mRNA from the AP-2 gene encodes a negative regulator of transcriptional activation by AP-2.

TL;DR: It is shown that constitutive AP-2B expression in PA-1 cells causes a retinoic acid-resistant phenotype, anchorage-independent growth in soft agar, and tumorigenicity in nude mice, in a fashion similar to transformation of these cells by oncogenes.