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Renata Cifkova

Researcher at First Faculty of Medicine, Charles University in Prague

Publications -  319
Citations -  91912

Renata Cifkova is an academic researcher from First Faculty of Medicine, Charles University in Prague. The author has contributed to research in topics: Population & Blood pressure. The author has an hindex of 68, co-authored 305 publications receiving 80868 citations. Previous affiliations of Renata Cifkova include Masaryk University & Czechoslovak Academy of Sciences.

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Blood pressure control and risk profile in poststroke survivors: a comparison with the general population.

TL;DR: A high prevalence and poor control of major cardiovascular risk factors in patients surviving their first-ever ischemic stroke is found, thus showing poor implementation of guidelines for secondary prevention in clinical practice.
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Serum Vitamin D Status, Vitamin D Receptor Polymorphism, and Glucose Homeostasis in Healthy Subjects.

TL;DR: Low vitamin D status was in virtually healthy subjects associated with decreased insulin sensitivity, namely in those with GG genotype of rs2228570 VDR polymorphism, and possible interaction with vitamin D receptor (VDR) polymorphism.
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The metabolic syndrome in hypertension: Diagnostic and therapeutic implications

TL;DR: Prevalence is higher than in the general population and the metabolic syndrome can be found in as many as one third of patients and a high prevalence of hypertension-induced target-organ damage and a poor prognostic value has been described.
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Why is the treatment of hypertension in pregnancy still so difficult

TL;DR: Sufficient data regarding treatment of hypertension in pregnancy are lacking as pharmaceutical companies have been reluctant to test drugs in this small market with a high potential of litigation and Pharmaceutical companies are not willing to take any risk and, therefore, no data are available for most of the antihypertensive drugs marketed over the last 20 years.
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Blood pressure, endothelial function and circulating endothelin concentrations in liver transplant recipients.

TL;DR: Results show increased blood pressure with suppressed circadian blood pressure variability in liver graft recipients 6 weeks after transplant and no change in endothelial function and plasma endothelin concentrations, which cannot be explained by endothelial dysfunction.