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Richard Stevens

Researcher at University of Oxford

Publications -  201
Citations -  31066

Richard Stevens is an academic researcher from University of Oxford. The author has contributed to research in topics: Type 2 diabetes & Diabetes mellitus. The author has an hindex of 44, co-authored 188 publications receiving 28480 citations. Previous affiliations of Richard Stevens include National Institute for Health Research & Cancer Epidemiology Unit.

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Intensive blood-glucose control with sulphonylureas or insulin compared with conventional treatment and risk of complications in patients with type 2 diabetes (UKPDS 33)

R C Turner, +398 more
- 12 Sep 1998 - 
TL;DR: In this article, the effects of intensive blood-glucose control with either sulphonylurea or insulin and conventional treatment on the risk of microvascular and macrovascular complications in patients with type 2 diabetes in a randomised controlled trial were compared.
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Development and progression of nephropathy in type 2 diabetes: the United Kingdom Prospective Diabetes Study (UKPDS 64).

TL;DR: Relatively fewer patients with type 2 diabetes develop macroalbuminuria, but in those who do, the death rate exceeds the rate of progression to worse nephropathy, and risk of death from all-causes or cardiovascular disease is high.
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The UKPDS risk engine: a model for the risk of coronary heart disease in Type II diabetes (UKPDS 56)

TL;DR: The model is diabetes-specific and incorporates glycaemia, systolic blood pressure and lipid levels as risk factors, in addition to age, sex, ethnic group, smoking status and time since diagnosis of diabetes, which provides the estimates ofCHD risk required by current guidelines for the primary prevention of CHD in Type II diabetes.
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Normal ranges of heart rate and respiratory rate in children from birth to 18 years of age: a systematic review of observational studies

TL;DR: The authors' evidence-based centile charts for children from birth to 18 years should help clinicians to update clinical and resuscitation guidelines and show decline in respiratory rate fromBirth to early adolescence.