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Robert J. Crawford

Researcher at University of Melbourne

Publications -  20
Citations -  1952

Robert J. Crawford is an academic researcher from University of Melbourne. The author has contributed to research in topics: Gene & Complementary DNA. The author has an hindex of 16, co-authored 20 publications receiving 1928 citations.

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Mouse prepro-epidermal growth factor synthesis by the kidney and other tissues

TL;DR: It is shown here that several mouse tissues contain preproEGF mRNA and that it is synthesized mainly in the distal tubules of the kidney, which does not seem to be processed to EGF or other peptides in this tissue.
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Cloning of cDNA for a stylar glycoprotein associated with expression of self-incompatibility in Nicotiana alata

TL;DR: A complementary DNA clone encoding a style protein of Nicotiana alata which segregates with the S2 allele has now been sequenced and shows that the S-allele-associated style components in different genotypes of N. alata and in Lycopersicon peruvianum are homologous.
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Structure of a genomic clone encoding biologically active human relaxin

TL;DR: Synthesis of biologically active relaxin has shown that the novel gene structure described herein codes for an authentic human relaxin, believed to be the first successful synthesis of a biologically active hormone whose structure was predicted solely from the structure of a genomic clone.
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Relaxin gene expression in human ovaries and the predicted structure of a human preprorelaxin by analysis of cDNA clones.

TL;DR: Evidence is presented that the human genome possesses an additional relaxin‐related gene (designated human relaxin gene H2) which appears to be selectively expressed in the ovary during pregnancy and which encodes an authentic human Relaxin.
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α-Inhibin Gene Expression Occurs in the Ovine Adrenal Cortex, and is Regulated by Adrenocorticotropin

TL;DR: Dot blot analysis showed that the level of alpha mRNA within the adrenal is influenced by ACTH, one of the major regulators of adrenal cortex function, implying that there are inhibin-related peptides not directly associated with the gonads.