R
Robert P. Erickson
Researcher at University of Arizona
Publications - 311
Citations - 13458
Robert P. Erickson is an academic researcher from University of Arizona. The author has contributed to research in topics: Gene & Gene expression. The author has an hindex of 52, co-authored 304 publications receiving 12801 citations. Previous affiliations of Robert P. Erickson include Lincoln's Inn & University of California, San Francisco.
Papers
More filters
Journal ArticleDOI
A Polymorphism* in the 5 ′ Flanking Region of the CD14 Gene Is Associated with Circulating Soluble CD14 Levels and with Total Serum Immunoglobulin E
Mauro Baldini,I. Carla Lohman,Marilyn Halonen,Robert P. Erickson,Patrick G. Holt,Fernando D. Martinez +5 more
TL;DR: It is concluded that CD14/-159 plays a significant role in regulating serum sCD14 levels and total serum IgE levels.
Journal ArticleDOI
Association between genetic polymorphisms of the beta2-adrenoceptor and response to albuterol in children with and without a history of wheezing.
TL;DR: Genotyped 269 children who were participants in a longitudinal study of asthma and marked linkage disequilibrium between the beta2AR-27 polymorphism and response to albuterol was found, which may explain some of the variability in response to therapeutic doses of al buterol in children.
Journal ArticleDOI
Mutations in FOXC2 (MFH-1), a forkhead family transcription factor, are responsible for the hereditary lymphedema-distichiasis syndrome.
Jianming Fang,Susan L. Dagenais,Robert P. Erickson,Martin F. Arlt,Michael W. Glynn,Jerome L. Gorski,Laurie H. Seaver,Thomas W. Glover +7 more
TL;DR: FoxC2 represents the second known gene to result in hereditary lymphedema, and LD is only the second hereditary disorder known to be caused by a mutation in a forkhead-family gene, and FOXC2 haploinsufficiency results in LD.
Journal ArticleDOI
The absence of mitochondrial thioredoxin 2 causes massive apoptosis, exencephaly, and early embryonic lethality in homozygous mice.
TL;DR: The results show that the mitochondrial redox protein Trx-2 is required for normal development of the mouse embryo and for actively respiring cells.