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Robert S. Gibson

Researcher at University of Virginia

Publications -  75
Citations -  6454

Robert S. Gibson is an academic researcher from University of Virginia. The author has contributed to research in topics: Myocardial infarction & Infarction. The author has an hindex of 38, co-authored 75 publications receiving 6400 citations. Previous affiliations of Robert S. Gibson include Brigham and Women's Hospital & University of Miami.

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Prediction of cardiac events after uncomplicated myocardial infarction: a prospective study comparing predischarge exercise thallium-201 scintigraphy and coronary angiography.

TL;DR: Submaximal exercise 200T1 scintigraphy can distinguish high- and low-risk groups after uncomplicated acute myocardial infarction before hospital discharge and 201T1 defects in more than one discrete vascular region, presence of delayed redistribution, or increased lung thallium uptake are more sensitive predictors of subsequent cardiac events than ST segment depression, angina, or extent of angiographic disease.
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Diltiazem and reinfarction in patients with non-Q-wave myocardial infarction. Results of a double-blind, randomized, multicenter trial.

TL;DR: It is concluded that diltiazem was effective in preventing early reinfarction and severe angina after non-Q-wave infarction and that it was also safe and generally well tolerated.
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Influence of race, sex, and age on management of unstable angina and non-Q-wave myocardial infarction: The TIMI III registry.

TL;DR: Among patients presenting with acute ischemic chest pain without persistent ST-segment elevation, blacks appeared to have less severe coronary disease, received revascularization less frequently, and had less recurrent ischemia compared with nonblacks, which suggests that more aggressive strategies should be directed to those patients with the greatest likelihood of adverse outcomes.
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Prospective assessment of regional myocardial perfusion before and after coronary revascularization surgery by Quantitative Thallium-201 scintigraphy

TL;DR: The preoperative distinction between viable and nonviable myocardium can be reasonably established by quantitating the amount of persistent reduction in thallium uptake and correlating this with preoperative wall motion.