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Robin F.H. Cash

Researcher at University of Melbourne

Publications -  60
Citations -  1801

Robin F.H. Cash is an academic researcher from University of Melbourne. The author has contributed to research in topics: Transcranial magnetic stimulation & Dorsolateral prefrontal cortex. The author has an hindex of 21, co-authored 55 publications receiving 1043 citations. Previous affiliations of Robin F.H. Cash include Monash University & University of Western Australia.

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Using Brain Imaging to Improve Spatial Targeting of Transcranial Magnetic Stimulation for Depression.

TL;DR: How neuroimaging is being used to identify a more focal therapeutic target for depression is reviewed and recent studies showing that more effective TMS targets in the frontal cortex are functionally connected to deep limbic regions such as the subgenual cingulate cortex are highlighted.
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Characterization of Glutamatergic and GABA A -Mediated Neurotransmission in Motor and Dorsolateral Prefrontal Cortex Using Paired-Pulse TMS–EEG

TL;DR: The data suggest that SICI and ICF can be recorded using TMS–EEG in DLPFC providing noninvasive measures of glutamatergic and GABAA receptor-mediated neurotransmission, which may facilitate future research attempting to ascertain the role of these neurotransmitters in the pathophysiology and treatment of neurological and psychiatric disorders.
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Functional Magnetic Resonance Imaging-Guided Personalization of Transcranial Magnetic Stimulation Treatment for Depression.

TL;DR: The authors assesses whether treatment response for depression could be improved via a single one-site-fits-all dorsolateral prefrontal cortex target, representing the group average optimal site of subgenual cingulate cortex functional connectivity, or whether target site personalization is necessary.
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Late cortical disinhibition in human motor cortex: a triple-pulse transcranial magnetic stimulation study.

TL;DR: TMS-induced LICI is associated with a period of disinhibited that is at first masked by LICI, but that outlasts LICI and gives rise to a period during which disinhibition predominates and net excitability is raised.