R
Roger A. Schultz
Researcher at University of Texas Southwestern Medical Center
Publications - 57
Citations - 26443
Roger A. Schultz is an academic researcher from University of Texas Southwestern Medical Center. The author has contributed to research in topics: Gene & Gene mapping. The author has an hindex of 27, co-authored 57 publications receiving 24988 citations. Previous affiliations of Roger A. Schultz include University of Texas at Dallas.
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Journal ArticleDOI
Genomic Organization, Chromosomal Localization, and Expression of the Murine Thromboxane Synthase Gene (Tbxas1) ☆
TL;DR: These studies provide the genetic tools and information for TS research in mice, which should expedite understanding of the genetic contribution of TS in normal physiology as well as in disease states.
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“Proximal-type” and Classic Epithelioid Sarcomas Represent a Clinicopathologic Continuum: Case Report
Dinesh Rakheja,Dinesh Rakheja,Kathleen S. Wilson,John J. Meehan,Roger A. Schultz,Ana M. Gomez,Ana M. Gomez +6 more
TL;DR: It is suggested that the proximal-type and the classic epithelioid sarcomas are not distinct entities but represent a continuum.
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Identification and molecular confirmation of a small chromosome 10q duplication [dir dup(10)(q24.2→q24.3)] Inherited from a mother mosaic for the abnormality
Vijay S. Tonk,Vijay S. Tonk,Nancy R. Schneider,Mauricio R. Delgado,Jen I. Mao,Roger A. Schultz +5 more
TL;DR: Fluorescence in situ hybridization approaches, including total chromosome painting and the use of regional specific cosmid probes, were used to confirm the chromosome 10q origin of the duplicated material.
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Upregulation of TRAG3 gene in urothelial carcinoma of the bladder
Jose Antonio Karam,Sandra Huang,Jinhai Fan,Jinhai Fan,Jennifer Stanfield,Roger A. Schultz,Rey Chen Pong,Xiankai Sun,Ralph P. Mason,Xian Jin Xie,Gang Niu,Xiaoyuan Chen,Eugene P. Frenkel,Arthur I. Sagalowsky,Jer Tsong Hsieh +14 more
TL;DR: Knowing the status of TRAG3 expression could help clinicians tailor treatment to a particular patient population that could benefit from treatment, while allocating patients with resistant tumors to new experimental therapies.
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Novel assay for Roberts syndrome assigns variable phenotypes to one complementation group
Lisa D. McDaniel,Lisa D. McDaniel,Robyn Prueitt,Lori C. Probst,Kathleen S. Wilson,Darrell J. Tomkins,Golder N. Wilson,Roger A. Schultz,Roger A. Schultz +8 more
TL;DR: It is demonstrated that a single complementation group defines RS patients with heterochromatic splaying regardless of clinical severity, and a permanent cell line established from a new RS patient with a more severe phenotype is established.