R
Rosa Bonaventura
Researcher at National Research Council
Publications - 38
Citations - 1341
Rosa Bonaventura is an academic researcher from National Research Council. The author has contributed to research in topics: Paracentrotus lividus & Sea urchin. The author has an hindex of 20, co-authored 35 publications receiving 1153 citations.
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Journal ArticleDOI
Cellular and biochemical responses to environmental and experimentally induced stress in sea urchin coelomocytes.
TL;DR: The present study confirms the immunological function of sea urchin coelomocytes, as indicated by the upregulation of the hsp70 molecular marker, and suggests that sea urchesin coELomocytes can be utilized as sensitive bio-indicators of environmental stress.
Journal ArticleDOI
Cadmium induces the expression of specific stress proteins in sea urchin embryos.
TL;DR: Sea urchin embryos represent a simple though significant model system to test how specific stress can simultaneously affect development and protein expression, and the effects of time-dependent continuous exposure to subacute/sublethal cadmium concentrations are studied.
Journal ArticleDOI
UVB radiation prevents skeleton growth and stimulates the expression of stress markers in sea urchin embryos.
TL;DR: The morphological effects observed 1, 24, and 48 h after exposure were correlated with a dose-dependent increase in the level and in the activation of two recognized stress markers consistent with their role in mediating cellular response to stress and suggesting a function in embryo survival.
Book ChapterDOI
Monitoring chemical and physical stress using sea urchin immune cells.
Valeria Matranga,Annalisa Pinsino,Monica Celi,A. Natoli,Rosa Bonaventura,Heinz-Christoph Schröder,W. E. G. Müller +6 more
TL;DR: This chapter briefly reviews the important features of coelomocytes and describes studies on their use in the laboratory and in the field for the assessment of chemical and physical pollution of the sea.
Journal ArticleDOI
Titanium dioxide nanoparticles stimulate sea urchin immune cell phagocytic activity involving TLR/p38 MAPK-mediated signalling pathway
Annalisa Pinsino,Roberta Russo,Rosa Bonaventura,Andrea Brunelli,Antonio Marcomini,Valeria Matranga +5 more
TL;DR: It is indicated that TiO2NPs influence the signal transduction downstream targets of p38 MAPK without eliciting an inflammatory response or other harmful effects on biological functions.