R
Rosine De Paepe
Researcher at University of Paris-Sud
Publications - 44
Citations - 3546
Rosine De Paepe is an academic researcher from University of Paris-Sud. The author has contributed to research in topics: Nicotiana sylvestris & Mitochondrion. The author has an hindex of 27, co-authored 43 publications receiving 3337 citations. Previous affiliations of Rosine De Paepe include Centre national de la recherche scientifique & University of Paris.
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Journal ArticleDOI
Leaf Mitochondria Modulate Whole Cell Redox Homeostasis, Set Antioxidant Capacity, and Determine Stress Resistance through Altered Signaling and Diurnal Regulation
Christelle Dutilleul,Marie Garmier,Graham Noctor,Chantal Mathieu,P. Chetrit,Christine H. Foyer,Rosine De Paepe +6 more
TL;DR: Loss of complex I function reveals effective antioxidant crosstalk and acclimation between the mitochondria and other organelles to maintain whole cell redox balance and is associated with higher tolerance to ozone and Tobacco mosaic virus.
Journal ArticleDOI
Mitochondrial redox biology and homeostasis in plants
TL;DR: In this review, mitochondrial redox metabolism is discussed in relation to the integrated cellular energy and redox function that controls plant cell biology and fate.
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Functional Mitochondrial Complex I Is Required by Tobacco Leaves for Optimal Photosynthetic Performance in Photorespiratory Conditions and during Transients
Christelle Dutilleul,Simon P. Driscoll,Gabriel Cornic,Rosine De Paepe,Christine H. Foyer,Graham Noctor +5 more
TL;DR: It is concluded that Mitochondrial complex I is required for optimal photosynthetic performance, despite the operation of alternative dehydrogenases in CMSII; andcomplex I is necessary to avoid redox disruption of photosynthesis in conditions where leaf mitochondria must oxidize both respiratory and photorespiratory substrates simultaneously.
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The nucleo-mitochondrial conflict in cytoplasmic male sterilities revisited.
TL;DR: The model of nuclear-mitochondrial conflict is re-examine in the light of recent experimental results from naturally occurring CMS, alloplasmic CMS (appearing after interspecific crosses resulting from the association of nuclear and cytoplasmic genomes from different species), and CMS plants obtained in the laboratory and carrying mitochondrial mutations.
Journal ArticleDOI
Lack of mitochondrial and nuclear-encoded subunits of complex I and alteration of the respiratory chain in Nicotiana sylvestris mitochondrial deletion mutants
Sophie Gutierres,Mohammed Sabar,Christine Lelandais,P. Chetrit,Philippe Diolez,Hervé Degand,Marc Boutry,Fernand Vedel,Yaroslav de Kouchkovsky,Rosine De Paepe +9 more
TL;DR: The capacity of the cyanide-resistant alternative oxidase pathway was enhanced in CMS, and higher amounts of enzyme were evidenced by immunodetection, suggesting respiratory compensation mechanisms involving additional NADH dehydrogenases to complex I.